Anti-atherogenic effect of BHB-TZD having inhibitory activities on cyclooxygenase and 5-lipoxygenase in hyperlipidemic mice

Atherosclerosis. 2010 Sep;212(1):146-52. doi: 10.1016/j.atherosclerosis.2010.05.003. Epub 2010 May 11.

Abstract

Cyclooxygenase (COX) and 5-lipoxygenase (5-LOX), which play pivotal roles in atherogenesis, have been reported to be involved in plaque stability. Licofelone, a dual COX and 5-LOX inhibitor, has been reported to possess anti-atherogenic effect in rabbit atherosclerosis model. We therefore investigated the anti-atherogenic effect of BHB-TZD [5-(3,5-di-tert-butyl-4-hydroxybenzylidene)thiazolidin-2,4-dione], a dual COX and 5-LOX inhibitor, in low density lipoprotein receptor null (LDLR-/-) mice. Fifteen LDLR-/- mice were fed a western diet (control group), whereas 15 were fed a western diet plus 0.1% (w/w) BHB-TZD (BHB-TZD group). After 8 weeks, the BHB-TZD group had markedly lower serum levels of leukotriene B(4) and prostaglandin E(2) than the control group. Interestingly, BHB-TZD treatment also reduced plasma triglyceride level without significant changes in total cholesterol and HDL levels. Compared with control mice, BHB-TZD fed mice had 52% fewer fatty streak lesions in the aortic sinus, as well as fewer initial lesions in the aortic arch. Macrophage infiltration into the lesions was 40% lower, and collagen and smooth muscle cells were increased by 102% and 96%, respectively, in the BHB-TZD group compared with the control group. In addition, aortic expression of proatherogenic molecules including TNF-alpha, IL-1beta, IL-6, MCP-1 and VCAM-1, was lower in the BHB-TZD group than the control group. BHB-TZD treatment also reduced MMP-2 and MMP-9 expressions in aorta. In conclusion, BHB-TZD effectively attenuated atherosclerosis in mouse model, suggesting its therapeutic potential for atherosclerosis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Aorta, Thoracic / drug effects
  • Aorta, Thoracic / enzymology
  • Aorta, Thoracic / pathology
  • Aortic Diseases / enzymology
  • Aortic Diseases / genetics
  • Aortic Diseases / pathology
  • Aortic Diseases / prevention & control*
  • Arachidonate 5-Lipoxygenase / metabolism*
  • Atherosclerosis / enzymology
  • Atherosclerosis / genetics
  • Atherosclerosis / prevention & control*
  • Cholesterol / blood
  • Cyclooxygenase Inhibitors / pharmacology*
  • Dinoprostone / blood
  • Disease Models, Animal
  • Gene Expression Regulation / drug effects
  • Hyperlipidemias / drug therapy*
  • Hyperlipidemias / enzymology
  • Hyperlipidemias / genetics
  • Inflammation Mediators / metabolism
  • Leukotriene B4 / blood
  • Lipoproteins, HDL / blood
  • Lipoxygenase Inhibitors / pharmacology*
  • Macrophages / drug effects
  • Macrophages / enzymology
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Myocytes, Smooth Muscle / drug effects
  • Myocytes, Smooth Muscle / enzymology
  • Prostaglandin-Endoperoxide Synthases / metabolism*
  • RNA, Messenger / metabolism
  • Receptors, LDL / deficiency
  • Receptors, LDL / genetics
  • Thiazolidinediones / pharmacology*
  • Time Factors
  • Triglycerides / blood

Substances

  • 5-(3,5-di-tert-butyl-4-hydroxybenzylidene)thiazolidine-2,4-dione
  • Cyclooxygenase Inhibitors
  • Inflammation Mediators
  • Lipoproteins, HDL
  • Lipoxygenase Inhibitors
  • RNA, Messenger
  • Receptors, LDL
  • Thiazolidinediones
  • Triglycerides
  • Leukotriene B4
  • Cholesterol
  • Arachidonate 5-Lipoxygenase
  • Prostaglandin-Endoperoxide Synthases
  • Dinoprostone