Commensal microflora and interferon-gamma promote steady-state interleukin-7 production in vivo

Eur J Immunol. 2010 Sep;40(9):2391-400. doi: 10.1002/eji.201040441.

Abstract

IL-7 is a major regulator of lymphocyte homeostasis; however, little is known about the mechanisms that regulate IL-7 production. To study Il7 gene regulation in vivo, we generated a novel IL-7-reporter mouse, which allows the non-invasive quantification of Il7 gene activity in live mice and, additionally, the simultaneous activation/inactivation of target genes in IL-7-producing cells. With these IL-7-reporter mice, we identify thymus, skin and intestine as major sources of IL-7 in vivo. Importantly, we show that IFN-gamma and the commensal microflora promote steady-state IL-7 production in the intestine. Furthermore, we demonstrate that the blockade of IFN-gamma signaling in intestinal epithelial cells strongly reduces their IFN-gamma-driven IL-7 production. In summary, our data suggest a feedback loop in which commensal bacteria drive IFN-gamma production by lymphocytes, which in turn promotes epithelial cell IL-7 production and the survival of IL-7-dependent lymphocytes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Anti-Bacterial Agents / administration & dosage
  • Cell Line, Tumor
  • Cell Survival / drug effects
  • Cell Survival / genetics
  • Cell Survival / immunology
  • Chromosomes, Artificial, Bacterial
  • Dexamethasone / pharmacology
  • Feedback, Physiological
  • Gene Expression Regulation / drug effects
  • Gene Expression Regulation / immunology
  • Interferon-gamma / genetics
  • Interferon-gamma / immunology
  • Interferon-gamma / metabolism*
  • Interleukin-7 / genetics
  • Interleukin-7 / immunology
  • Interleukin-7 / metabolism*
  • Intestinal Mucosa / drug effects
  • Intestinal Mucosa / immunology
  • Intestinal Mucosa / metabolism*
  • Intestinal Mucosa / pathology
  • Intestines / drug effects
  • Intestines / microbiology
  • Intestines / pathology
  • Luminescent Proteins / genetics
  • Luminescent Proteins / metabolism
  • Metagenome / drug effects
  • Metagenome / immunology
  • Mice
  • Mice, Transgenic
  • Signal Transduction / drug effects
  • Signal Transduction / genetics
  • T-Lymphocytes / immunology
  • T-Lymphocytes / metabolism
  • T-Lymphocytes / pathology

Substances

  • Anti-Bacterial Agents
  • Interleukin-7
  • Luminescent Proteins
  • Dexamethasone
  • Interferon-gamma