Decreasing expression of the interleukin-13 receptor IL-13Ralpha2 in treated recurrent malignant gliomas

Neurol Med Chir (Tokyo). 2010;50(8):617-21. doi: 10.2176/nmc.50.617.

Abstract

The IL-13Ralpha2 gene encodes for a 65 kDa protein that forms one of the subunits of the interleukin-13 (IL-13) receptor. This gene is highly expressed in various types of human tumors including malignant gliomas. The expression level of IL-13Ralpha2 was examined in a total of 45 tissue samples of anaplastic astrocytomas (AAs) World Health Organization (WHO) grade III, glioblastomas (GBMs) WHO grade IV, and first-recurrent glioblastomas (frGBMs) after treatment with radiation and chemotherapy. IL-13Ralpha2 expression was detected by semiquantitative reverse transcription real-time polymerase chain reaction (PCR) using ABI PRISM 7700 and Qiagen QuantiTect SYBR Green PCR kits. The expression level of IL-13Ralpha2 (15 fold) was significantly reduced in frGBMs compared to the primary GBMs (p = 0.014), and significantly reduced by more than 15 fold (p = 0.003) in all untreated malignant astrocytomas (AAs and GBMs) compared with treated frGBMs. Expression of IL-13Ralpha2 seems to be lower in frGBMs compared to GBMs. The promising antitumor effect of IL-13 cytotoxin could be greatly reduced in frGBM or only achievable with higher amounts of cytotoxin, due to the significantly lower expression of the cytotoxin's target structure.

Publication types

  • Comparative Study

MeSH terms

  • Actins / genetics
  • Actins / metabolism
  • Adult
  • Aged
  • Antineoplastic Combined Chemotherapy Protocols / therapeutic use
  • Astrocytoma / metabolism*
  • Astrocytoma / pathology
  • Astrocytoma / therapy
  • Brain Neoplasms / metabolism*
  • Brain Neoplasms / pathology
  • Brain Neoplasms / therapy
  • Exotoxins / therapeutic use
  • Female
  • Gene Expression Regulation, Neoplastic / drug effects
  • Glioblastoma / metabolism*
  • Glioblastoma / pathology
  • Glioblastoma / therapy
  • Humans
  • Immunotoxins / therapeutic use
  • Interleukin-13 / therapeutic use
  • Interleukin-13 Receptor alpha2 Subunit / genetics
  • Interleukin-13 Receptor alpha2 Subunit / metabolism*
  • Male
  • Middle Aged
  • Neoplasm Recurrence, Local / metabolism*
  • Neoplasm Recurrence, Local / pathology
  • Nimustine / administration & dosage
  • RNA / analysis
  • Recombinant Fusion Proteins
  • Teniposide / administration & dosage
  • Young Adult

Substances

  • Actins
  • Exotoxins
  • IL13-PE38
  • Immunotoxins
  • Interleukin-13
  • Interleukin-13 Receptor alpha2 Subunit
  • Recombinant Fusion Proteins
  • Nimustine
  • RNA
  • Teniposide