Abstract
Phosphatidylinositol-4,5-bisphosphate [PI(4,5)P(2)] is a membrane lipid involved in several signaling pathways. However, the role of this lipid in the regulation of synapse growth is ill-defined. Here we identify PI(4,5)P(2) as a gatekeeper of neuromuscular junction (NMJ) size. We show that PI(4,5)P(2) levels in neurons are critical in restricting synaptic growth by localizing and activating presynaptic Wiscott-Aldrich syndrome protein/WASP (WSP). This function of WSP is independent of bone morphogenetic protein (BMP) signaling but is dependent on Tweek, a neuronally expressed protein. Loss of PI(4,5)P(2)-mediated WSP activation results in increased formation of membrane-organizing extension spike protein (Moesin)-GFP patches that concentrate at sites of bouton growth. Based on pharmacological and genetic studies, Moesin patches mark polymerized actin accumulations and correlate well with NMJ size. We propose a model in which PI(4,5)P(2)- and WSP-mediated signaling at presynaptic termini controls actin-dependent synapse growth in a pathway at least in part in parallel to synaptic BMP signaling.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Animals
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Bone Morphogenetic Proteins / genetics
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Bone Morphogenetic Proteins / metabolism*
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Drosophila Proteins / genetics
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Drosophila Proteins / metabolism
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Drosophila melanogaster / anatomy & histology
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Drosophila melanogaster / physiology
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Microfilament Proteins / genetics
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Microfilament Proteins / metabolism
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Nerve Tissue Proteins / genetics
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Nerve Tissue Proteins / metabolism
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Neuromuscular Junction / physiology
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Neuromuscular Junction / ultrastructure
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Patch-Clamp Techniques
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Phosphatidylinositol 4,5-Diphosphate / metabolism*
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Presynaptic Terminals / metabolism
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Presynaptic Terminals / ultrastructure
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Receptors, Cell Surface / genetics
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Receptors, Cell Surface / metabolism
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Recombinant Fusion Proteins / genetics
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Recombinant Fusion Proteins / metabolism
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Signal Transduction / physiology*
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Synapses / physiology*
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Synapses / ultrastructure
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Wiskott-Aldrich Syndrome Protein / genetics
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Wiskott-Aldrich Syndrome Protein / metabolism*
Substances
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Bone Morphogenetic Proteins
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Drosophila Proteins
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Microfilament Proteins
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Nerve Tissue Proteins
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Phosphatidylinositol 4,5-Diphosphate
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Receptors, Cell Surface
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Recombinant Fusion Proteins
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Wiskott-Aldrich Syndrome Protein
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nwk protein, Drosophila
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wit protein, Drosophila
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moesin