Abstract
Among the enzymes involved in the life cycle of HCV, the non-structural protein NS3, with its double function of protease and NTPase/helicase, is essential for the virus replication. Exploiting our previous knowledge in the development of nucleotide-mimicking NS3 helicase (NS3h) inhibitors endowed with key structural and electronic features necessary for an optimal ligand-enzyme interaction, we developed the tetrahydroacridinyl derivative 3a as the most potent NS3h competitive inhibitor reported to date (HCV NS3h K(i)=20 nM).
Copyright © 2010 Elsevier Ltd. All rights reserved.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Binding, Competitive / drug effects
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Drug Discovery*
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Hepacivirus / enzymology*
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Hydrazines / chemical synthesis
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Hydrazines / chemistry*
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Hydrazines / pharmacology*
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Protein Binding
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Pyrazines / chemical synthesis
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Pyrazines / chemistry*
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Pyrazines / pharmacology*
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Quinolines / chemical synthesis
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Quinolines / chemistry*
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Quinolines / pharmacology*
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Viral Nonstructural Proteins / antagonists & inhibitors*
Substances
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Hydrazines
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NS3 protein, hepatitis C virus
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Pyrazines
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QU663 compound
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Quinolines
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Viral Nonstructural Proteins