Abstract
Helicobacter (Campylobacter) pylori was found to be sensitive to the toxic effects of an unsaturated fatty acid (arachidonic acid). Data are presented that support the hypothesis that exogenous catalase added to basal media enhances the growth of H. pylori by preventing the formation of toxic peroxidation products from long-chain unsaturated fatty acids.
Publication types
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Research Support, Non-U.S. Gov't
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Research Support, U.S. Gov't, Non-P.H.S.
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Research Support, U.S. Gov't, P.H.S.
MeSH terms
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Arachidonic Acid
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Arachidonic Acids / pharmacology*
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Campylobacter / drug effects*
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Campylobacter / growth & development
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Campylobacter / metabolism
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Catalase / metabolism
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Catalase / pharmacology
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Culture Media
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Drug Resistance, Microbial
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Fatty Acids, Unsaturated / metabolism
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Humans
Substances
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Arachidonic Acids
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Culture Media
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Fatty Acids, Unsaturated
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Arachidonic Acid
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Catalase