Apolipoprotein AIMilano. Disulfide-linked dimers increase high density lipoprotein stability and hinder particle interconversion in carrier plasma

J Biol Chem. 1990 Jul 25;265(21):12224-31.

Abstract

The in vitro metabolism of high density lipoproteins (HDL) in carriers of the apolipoprotein AIMilano (apoAIM) mutant was investigated during incubation of whole plasma and isolated lipoprotein fractions. A reduced cholesterol esterification (16.5 versus 25.0% for controls) and a decreased exchange of lipids between HDL and lower density lipoproteins was observed during incubation (6 h at 37 degrees C) of AIM plasma. Control HDL3 were converted to larger, faster-floating HDL particles, whereas only a fraction of AIM HDL3 followed the same pathway. Incubations were also carried out by mixing HDL3 from controls and AIM carriers with a lipoprotein-depleted plasma fraction in the presence of triglyceride-rich particles isolated from Intralipid. AIM HDL3 again showed a reduced capacity for lipid exchange; some HDL3 particles followed a "normal" conversion to faster-floating, larger HDL, whereas the small AIM HDL3 were not modified, indicating that AIM HDL3 are a mixture of metabolically functional and nonfunctional particles. Following transformation of the apoAIM homo- and heterodimers into their normal counterparts, i.e. monomeric apoAI and -AII, by reduction and carboxamidomethylation of AIM HDL3, the modified HDL3 behave like control HDL3 during incubation with lipoprotein-depleted plasma and triglyceride-rich particles. The presence of AIM dimers is most likely responsible for the increased HDL3 stability in the AIM carriers, indicating that apolipoprotein composition plays a major role in HDL particle interconversion.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Apolipoprotein A-I
  • Apolipoproteins A / genetics
  • Apolipoproteins A / physiology*
  • Carrier Proteins / metabolism
  • Cholesterol / blood
  • Cholesterol Esters / blood
  • Disulfides
  • Heterozygote
  • Humans
  • Lipoproteins, HDL / metabolism*
  • Mutation
  • Phosphatidylcholine-Sterol O-Acyltransferase / metabolism
  • Triglycerides / blood

Substances

  • Apolipoprotein A-I
  • Apolipoproteins A
  • Carrier Proteins
  • Cholesterol Esters
  • Disulfides
  • Lipoproteins, HDL
  • Triglycerides
  • lipid transfer protein
  • Cholesterol
  • Phosphatidylcholine-Sterol O-Acyltransferase