The relationship between interferon-γ gene polymorphism and acute kidney allograft rejection

Scand J Immunol. 2011 Apr;73(4):319-24. doi: 10.1111/j.1365-3083.2010.02506.x.

Abstract

Cytokine gene polymorphisms have been associated with modified gene expression and cytokine production. Gamma interferon (IFN-γ) plays an important role in the pathogenesis of kidney transplant rejection. This study evaluated the association between IFN-γ gene polymorphisms and the history of acute allograft rejection in 53 adult first-transplant recipients receiving cadaveric kidney grafts. They were followed up in a single centre until 2006, for a median time of 4 years after transplantation (1-22 years). IFN-γ gene polymorphisms +874 T/A (rs2430561) were determined by polymerase chain reaction (PCR). T/T high IFN-γ genotype was found in 12, intermediate T/A in 29 and low A/A in 12 patients. Twenty-six acute kidney rejection episodes were evidenced in 20 patients, of which none occurred in the 12 patients with low IFN-γ genotype A/A. Age, gender, number of HLA (human leukocyte antigen) mismatches, ABO blood groups, HLA, time after transplantation, creatinine clearance and immunosuppressive regimens were excluded as confounding factors associated with IFN-γ genotype distribution between rejectors and non-rejectors. IFN-γ gene polymorphisms could be an important risk factor for acute kidney transplant rejection, whereas the low A/A IFN-γ genotype could be protective against rejection.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • ABO Blood-Group System / immunology
  • Adult
  • Age Factors
  • Aged
  • Confounding Factors, Epidemiologic
  • Female
  • Genotype
  • Graft Rejection / epidemiology
  • Graft Rejection / genetics*
  • Graft Rejection / immunology*
  • HLA Antigens / immunology
  • Humans
  • Immunosuppression Therapy / methods
  • Interferon-gamma / genetics*
  • Interferon-gamma / immunology
  • Kidney Transplantation / immunology*
  • Male
  • Middle Aged
  • Polymorphism, Single Nucleotide / genetics*
  • Polymorphism, Single Nucleotide / immunology*
  • Risk Factors
  • Sex Factors
  • Tissue Donors
  • Transplantation, Homologous
  • Young Adult

Substances

  • ABO Blood-Group System
  • HLA Antigens
  • Interferon-gamma