Chemical enhancement of cisplatin cytotoxicity in a human ovarian and cervical cancer cell line

Gynecol Oncol. 1990 Sep;38(3):315-22. doi: 10.1016/0090-8258(90)90065-s.

Abstract

While many advances have been made in the chemotherapy of gynecologic cancers, treatment failures remain a major clinical problem. A growing understanding of the mechanisms of tumor cell resistance to antineoplastic drugs provides a framework for the development of chemotherapy regimens containing agents capable of modulating tumor response. Using a short-term ATP bioluminescence assay we studied the ability of two methylxanthines (caffeine, pentoxifylline) and an inhibitor of ADP-ribosyl transferase (3-aminobenzamide) to enhance cisplatin cytotoxicity in gynecologic cancer cell lines. Our findings of significantly enhanced cisplatin-induced cytotoxicity with two different analysis techniques confirms the effectiveness of these agents. These results may have future clinical significance.

MeSH terms

  • Adenosine Triphosphate / analysis
  • Benzamides
  • Caffeine / pharmacology
  • Cells, Cultured
  • Cisplatin / pharmacology*
  • Dose-Response Relationship, Drug
  • Drug Resistance
  • Drug Synergism
  • Female
  • Humans
  • In Vitro Techniques
  • Ovarian Neoplasms / drug therapy*
  • Pentoxifylline / pharmacology
  • Poly(ADP-ribose) Polymerases / pharmacology
  • Uterine Cervical Neoplasms / drug therapy*

Substances

  • Benzamides
  • Caffeine
  • 3-aminobenzamide
  • Adenosine Triphosphate
  • Poly(ADP-ribose) Polymerases
  • Cisplatin
  • Pentoxifylline