Upregulation of interferon-gamma-induced genes during prion infection

J Toxicol Environ Health A. 2011;74(2-4):146-53. doi: 10.1080/15287394.2011.529064.

Abstract

Global gene expression analysis allows for the identification of transcripts that are differentially regulated during a disease state. Many groups, including our own, have identified hundreds of genes differentially regulated in response to prion infection. Eleven transcripts, upregulated in the brains of prion-infected animals, which were classified in the literature as stimulated by the cytokine interferon-gamma (IFN-γ), were identified. This is intriguing, as IFN-γ has recently been detected in the brains of prion-infected animals. Quantitation of several genes, categorized as IFN-γ inducible, by quantitative real-time polymerase chain reaction (qRT-PCR) confirms that these transcripts are upregulated. Future approaches for delineating the role of IFN-γ-induced transcripts and their function in prion infection are described.

MeSH terms

  • Animals
  • Brain / metabolism
  • Interferon-gamma / genetics*
  • Interferon-gamma / physiology
  • Mice
  • Mice, Inbred C57BL
  • Oligonucleotide Array Sequence Analysis
  • Prion Diseases / genetics*
  • Prion Diseases / metabolism
  • RNA, Messenger / genetics
  • RNA, Messenger / isolation & purification
  • Reverse Transcriptase Polymerase Chain Reaction
  • Up-Regulation / genetics

Substances

  • RNA, Messenger
  • Interferon-gamma