Cytokine regulation of HLA on thyroid epithelial cells

Clin Exp Immunol. 1990 Dec;82(3):548-52. doi: 10.1111/j.1365-2249.1990.tb05488.x.

Abstract

The regulation of class I and class II HLA expression in human thyroid follicular cells was studied in vitro. Tumour necrosis factor-alpha (TNF-alpha) enhanced the expression of class I antigen on thyrocytes, but these cytokines had little effect on the expression of class II antigen. Interleukin-1 beta (IL-1 beta) and interleukin-6 (IL-6) did not affect class I and class II antigen expression. The combination of interferon-gamma (IFN-gamma) with TNF-alpha or IL-1 beta enhanced the induction of class I and class II antigens, compared with the effect of IFN-gamma alone. Neither class I nor class II expression was induced by IL-6 alone or in combination with IFN-gamma. These findings suggest that TNF-alpha and IL-1 beta may have an important role in inappropriate expression of HLA antigens on thyrocytes in thyroid gland.

MeSH terms

  • Cell Survival / drug effects
  • Cytokines / pharmacology*
  • Dose-Response Relationship, Drug
  • Drug Synergism
  • Epithelium / immunology
  • Flow Cytometry
  • Gene Expression Regulation
  • HLA Antigens / biosynthesis*
  • Histocompatibility Antigens Class I / biosynthesis
  • Histocompatibility Antigens Class II / biosynthesis
  • Humans
  • In Vitro Techniques
  • Interferon-gamma / pharmacology
  • Interleukin-1 / pharmacology
  • Interleukin-6 / pharmacology
  • Thyroid Gland / drug effects
  • Thyroid Gland / immunology*
  • Tumor Necrosis Factor-alpha / pharmacology

Substances

  • Cytokines
  • HLA Antigens
  • Histocompatibility Antigens Class I
  • Histocompatibility Antigens Class II
  • Interleukin-1
  • Interleukin-6
  • Tumor Necrosis Factor-alpha
  • Interferon-gamma