Evidence for release of prostaglandin F2 alpha in contractile response of guinea pig trachea to norepinephrine

Jpn J Pharmacol. 1990 Nov;54(3):330-2. doi: 10.1254/jjp.54.330.

Abstract

The concentration (10(-8)-10(-5) M)-dependent contractile response of norepinephrine was completely inhibited by cyclooxygenase inhibitors but not by a thromboxane synthetase inhibitor. The amount of prostaglandin F2 alpha was significantly increased in the presence of norepinephrine (10(-5) M). Norepinephrine increased the amount of prostaglandin D2, although not significantly. The norepinephrine-evoked prostaglandin release was inhibited by tolazoline (3 x 10(-5) M). These results suggest that norepinephrine evoked the release of prostaglandins, mainly prostaglandin F2 alpha to induce the contraction of the guinea pig trachea.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cyclooxygenase Inhibitors
  • Dinoprost / metabolism*
  • Guinea Pigs
  • In Vitro Techniques
  • Male
  • Muscle Relaxation / drug effects
  • Muscle, Smooth / drug effects
  • Muscle, Smooth / metabolism
  • Muscle, Smooth / physiology*
  • Norepinephrine / pharmacology*
  • Tolazoline / pharmacology
  • Trachea / drug effects

Substances

  • Cyclooxygenase Inhibitors
  • Dinoprost
  • Tolazoline
  • Norepinephrine