Functional and phenotypic modifications induced by IL-4, as single agent or in combination with IL-2, on PBMC preactivated in vivo by alpha-interferon + interleukin-2 therapy

Eur Cytokine Netw. 1990 Aug-Sep;1(3):141-7.

Abstract

The effect of human IL-4, used as a single agent or in combination with low or high dose IL-2, upon LAK-cell proliferation and activation has been tested on PBMC from patients treated with alpha 2-IFN and IL-2. Four days in vitro culture with IL-4 did not induce any LAK-cell activation; IL-4 induced the proliferation of CD3+ CD4+ T-cells, but decreased the percentage of NK cells in culture samples. When combined with high dose IL-2, IL-4 improved the recovery of MN cell without modification of T-cell subsets; however, IL-4 had no major effect on IL-2-induced NK or LAK cell activity. The combination of IL-4 and low dose IL-2 still significantly improved the total MN cell recovery but did not modify the distribution of T and NK lymphocytes; IL-4 inhibited low dose IL-2-induced NK and LAK cell activity, and increased the BL-esterase activity induced by high or low dose IL-2. The combination of IL-4 and IL-2 did not induce any large variation in the percentage of IL-2R (p55) expressing cells. In all tested conditions, IL-2R (p55) was mainly expressed on CD4+ T cells; less than 2% of the cells coexpressed the NK cell marker CD56 and IL-2R (p55). The effect of IL-4 upon IL-2-induced LAK cell expansion is thus very different on PBMC pre-activated in vivo by alpha IFN + IL-2 therapy than on PBMC pre-treated in vitro with IL-2.(ABSTRACT TRUNCATED AT 250 WORDS)

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Carcinoma, Renal Cell / immunology
  • Carcinoma, Renal Cell / therapy
  • Cells, Cultured
  • Drug Interactions
  • Humans
  • Immunologic Factors / therapeutic use
  • Interferon Type I / administration & dosage
  • Interferon Type I / pharmacology*
  • Interferon Type I / therapeutic use
  • Interleukin-2 / administration & dosage
  • Interleukin-2 / pharmacology*
  • Interleukin-2 / therapeutic use
  • Interleukin-4 / administration & dosage
  • Interleukin-4 / pharmacology*
  • Kidney Neoplasms / immunology
  • Kidney Neoplasms / therapy
  • Killer Cells, Lymphokine-Activated / drug effects*
  • Killer Cells, Natural / drug effects
  • Leukocytes, Mononuclear / drug effects*
  • Lymphocyte Activation / drug effects*
  • Middle Aged
  • Recombinant Proteins / pharmacology
  • Recombinant Proteins / therapeutic use
  • T-Lymphocyte Subsets / drug effects

Substances

  • Immunologic Factors
  • Interferon Type I
  • Interleukin-2
  • Recombinant Proteins
  • Interleukin-4