Characterization of mannose-binding lectin plasma levels and genetic polymorphisms in HIV-1-infected individuals

Rev Soc Bras Med Trop. 2011 Jan-Feb;44(1):1-3. doi: 10.1590/s0037-86822011000100001.

Abstract

Introduction: The present study investigated the association between mannose-binding lectin (MBL) gene polymorphism and serum levels with infection by HIV-1.

Methods: Blood samples (5 mL) were collected from 97 HIV-1-infected individuals resident in Belém, State of Pará, Brazil, who attended the Special Outpatient Unit for Infections and Parasitic Diseases (URE-DIPE). CD4+ T-lymphocyte count and plasma viral load were quantified. A 349bp fragment of exon 1 of the MBL was amplified via PCR, using genomic DNA extracted from controls and HIV-1-infected individuals, following established protocols. MBL plasma levels of the patients were quantified using an enzyme immunoassay kit.

Results: Two alleles were observed: MBL*O, with a frequency of 26.3% in HIV-1-infected individuals; and the wild allele MBL*A (73.7%). Similar frequencies were observed in the control group (p > 0.05). Genotype frequencies were distributed according to the Hardy-Weinberg equilibrium in both groups. Mean MBL plasma levels varied by genotype, with statistically significant differences between the AA and AO (p < 0.0001), and AA and OO (p < 0.001) genotypes, but not AO and OO (p = 0.17). Additionally, CD4+ T-lymphocytes and plasma viral load levels did not differ significantly by genotype (p > 0.05).

Conclusions: The results of this study do not support the hypothesis that MBL gene polymorphism or low plasma MBL concentrations might have a direct influence on HIV-1 infection, although a broader study involving a large number of patients is needed.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • CD4 Lymphocyte Count
  • Case-Control Studies
  • HIV Infections / blood*
  • HIV Infections / genetics
  • HIV Infections / virology
  • HIV-1 / genetics*
  • Humans
  • Mannose-Binding Lectin / blood*
  • Mannose-Binding Lectin / genetics*
  • Polymerase Chain Reaction
  • Polymorphism, Genetic / genetics*
  • Viral Load

Substances

  • Mannose-Binding Lectin