High-throughput screen of protein expression levels induced by cyclooxygenase-2 during influenza a virus infection

Clin Chim Acta. 2011 May 12;412(11-12):1081-5. doi: 10.1016/j.cca.2011.02.028. Epub 2011 Feb 24.

Abstract

Background: Detection of proteins productions and the functions during the influenza A virus infection, especially the proteins expression levels in the patients serum are emphasized in the research of host immune response to influenza virus infection. Protein microarray technology provides a high-throughput platform for efficient profiling of protein expression.

Methods: We investigated the expression levels of 507 immune-related proteins in the 85 serum of patients infected by influenza A virus H3N2 using an antibody array and validated these findings by infecting peripheral blood mononuclear cells (PBMCs) with A/HongKong/498/97 (H3N2) in vitro. Then we used selective inhibitor of proinflammatory factor cyclooxygenase-2 (COX-2) NS-398 to identify those immunomodulatory proteins regulated by the proinflammatory factor.

Results: In patients' serum, the expression levels of 138 proteins changed >2-fold in response to viral infection, including 102 that were upregulated and 36 that were downregulated. One-hundred six proteins were confirmed in PBMCs infected by H3N2. Of the 106 proteins involved in the immune response to influenza virus infection, 48 were regulated by COX-2.

Conclusions: Our findings identify the host proteins whose expression levels change in response to influenza virus infection and those involved in the proinflammatory factor COX-2-mediated inflammatory cascade.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cyclooxygenase 2 / metabolism*
  • Cyclooxygenase 2 Inhibitors / pharmacology
  • Cytokines / metabolism
  • Gene Expression Profiling*
  • Gene Expression Regulation, Enzymologic* / drug effects
  • Gene Expression Regulation, Enzymologic* / immunology
  • Humans
  • Immunologic Factors / metabolism
  • Influenza A Virus, H3N2 Subtype / pathogenicity*
  • Influenza, Human / blood
  • Influenza, Human / enzymology
  • Influenza, Human / immunology
  • Influenza, Human / metabolism*
  • Leukocytes, Mononuclear / virology
  • Nitrobenzenes / pharmacology
  • Protein Array Analysis*
  • Sulfonamides / pharmacology

Substances

  • Cyclooxygenase 2 Inhibitors
  • Cytokines
  • Immunologic Factors
  • Nitrobenzenes
  • Sulfonamides
  • N-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide
  • Cyclooxygenase 2