Hox genes define distinct progenitor sub-domains within the second heart field

Dev Biol. 2011 May 15;353(2):266-74. doi: 10.1016/j.ydbio.2011.02.029. Epub 2011 Mar 6.

Abstract

Much of the heart, including the atria, right ventricle and outflow tract (OFT) is derived from a progenitor cell population termed the second heart field (SHF) that contributes progressively to the embryonic heart during cardiac looping. Several studies have revealed anterior-posterior patterning of the SHF, since the anterior region (anterior heart field) contributes to right ventricular and OFT myocardium whereas the posterior region gives rise to the atria. We have previously shown that Retinoic Acid (RA) signal participates to this patterning. We now show that Hoxb1, Hoxa1, and Hoxa3, as downstream RA targets, are expressed in distinct sub-domains within the SHF. Our genetic lineage tracing analysis revealed that Hoxb1, Hoxa1 and Hoxa3-expressing cardiac progenitor cells contribute to both atria and the inferior wall of the OFT, which subsequently gives rise to myocardium at the base of pulmonary trunk. By contrast to Hoxb1(Cre), the contribution of Hoxa1-enhIII-Cre and Hoxa3(Cre)-labeled cells is restricted to the distal regions of the OFT suggesting that proximo-distal patterning of the OFT is related to SHF sub-domains characterized by combinatorial Hox genes expression. Manipulation of RA signaling pathways showed that RA is required for the correct deployment of Hox-expressing SHF cells. This report provides new insights into the regulatory gene network in SHF cells contributing to the atria and sub-pulmonary myocardium.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Aldehyde Oxidoreductases / deficiency
  • Aldehyde Oxidoreductases / genetics
  • Animals
  • Body Patterning / genetics
  • Body Patterning / physiology
  • Cell Lineage
  • Embryonic Stem Cells / cytology
  • Embryonic Stem Cells / metabolism
  • Female
  • Fetal Heart / cytology
  • Fetal Heart / embryology*
  • Fetal Heart / metabolism*
  • Gene Expression Regulation, Developmental
  • Gene Regulatory Networks
  • Genes, Homeobox*
  • Homeodomain Proteins / genetics
  • Homeodomain Proteins / metabolism
  • In Situ Hybridization
  • Male
  • Mice
  • Mice, Knockout
  • Mice, Transgenic
  • Models, Cardiovascular
  • Pregnancy
  • Signal Transduction
  • Transcription Factors / deficiency
  • Transcription Factors / genetics
  • Transcription Factors / metabolism
  • Tretinoin / metabolism

Substances

  • HOXB1 homeodomain protein
  • Homeodomain Proteins
  • Hoxa3 protein, mouse
  • Transcription Factors
  • homeobox A1 protein
  • Tretinoin
  • Aldehyde Oxidoreductases
  • RALDH2 protein, mouse