Insulin receptor activation in deletion 11q chronic lymphocytic leukemia

Clin Cancer Res. 2011 May 1;17(9):2605-7. doi: 10.1158/1078-0432.CCR-11-0295. Epub 2011 Mar 22.

Abstract

The chromosomal abnormalities characteristic of chronic lymphocytic leukemia (CLL) are well studied, but the mechanisms underlying their contribution to pathogenesis are only partially elucidated. Integrated genomic profiling, focused on deletion 11q, has identified elevated expression of the insulin receptor in a subgroup of CLLs and associated it with worse outcomes.

Publication types

  • Comment
  • Review

MeSH terms

  • Chromosome Deletion*
  • Chromosomes, Human, Pair 11* / genetics
  • Humans
  • Leukemia, Lymphocytic, Chronic, B-Cell / diagnosis
  • Leukemia, Lymphocytic, Chronic, B-Cell / genetics*
  • Leukemia, Lymphocytic, Chronic, B-Cell / metabolism
  • Models, Biological
  • Prognosis
  • Receptor, Insulin / metabolism*

Substances

  • Receptor, Insulin