Tumor necrosis factor alpha inhibitors as immunomodulatory antirejection agents after intestinal transplantation

Am J Transplant. 2011 May;11(5):1041-50. doi: 10.1111/j.1600-6143.2011.03497.x.

Abstract

We reported the successful administration of infliximab for late-onset OKT3-resistant rejection in two patients, who presented persistent ulcerative inflammation of the ileal graft after intestinal transplantation (ITX). Based on this experience, the present study demonstrated our long-term experience with infliximab for different types of rejection-related and inflammatory allograft alterations. Infliximab administration (5 mg/kg body weight (BW)) was initiated at a mean of 18.2 ± 14.1 months after transplantation. The number of administrations per patient averaged 8.4 ± 6.7. Repeat dosing was timed according to clinical signs and graft histology in addition to serum-levels of tumor necrosis factor alpha (TNFα), lipopolysaccharide binding protein (LBP) and C-reactive protein (CRP). Infliximab was successful in the following patients: patients with late-onset OKT3- and steroid-refractory rejection who presented persistent ulcerative alterations of the ileal graft (n = 5), patients with ulcerative ileitis/anastomositis, who did not show typical histological rejection signs (n = 2), and one patient with early-onset OKT3-resistant rejection. Infliximab was not successful in one patient with early-onset OKT3-resistant rejection that was accompanied by treatment-refractory humoral rejection. In conclusion, infliximab can expand therapeutic options for late-onset OKT3- and steroid-refractory rejection and chronic inflammatory graft alterations in intestinal allograft recipients.

MeSH terms

  • Acute-Phase Proteins / metabolism
  • Adult
  • Antibodies, Monoclonal / therapeutic use
  • Body Weight
  • C-Reactive Protein / metabolism
  • Carrier Proteins / metabolism
  • Female
  • Graft Rejection
  • Graft Survival
  • Humans
  • Immune System
  • Immunosuppressive Agents / therapeutic use*
  • Inflammation
  • Infliximab
  • Intestines / transplantation*
  • Male
  • Membrane Glycoproteins / metabolism
  • Steroids / pharmacology
  • Transplantation, Homologous
  • Treatment Outcome
  • Tumor Necrosis Factor-alpha / antagonists & inhibitors*

Substances

  • Acute-Phase Proteins
  • Antibodies, Monoclonal
  • Carrier Proteins
  • Immunosuppressive Agents
  • Membrane Glycoproteins
  • Steroids
  • Tumor Necrosis Factor-alpha
  • lipopolysaccharide-binding protein
  • C-Reactive Protein
  • Infliximab