Mechanisms of subcutaneous allergen immunotherapy

Immunol Allergy Clin North Am. 2011 May;31(2):175-90, vii-viii. doi: 10.1016/j.iac.2011.02.006.

Abstract

Allergen-specific immunotherapy (SIT) is the only curative approach in the treatment of allergic diseases defined up-to-date. Peripheral T-cell tolerance to allergens, the goal of successful allergen-SIT, is the primary mechanism in healthy immune responses to allergens. By repeated administration of increased doses of the causative allergen, allergen-SIT induces a state of immune tolerance to allergens through the constitution of T regulatory (Treg) cells, including allergen-specific interleukin (IL)-10-secreting Treg type 1 cells and CD4(+)CD25(+)Treg cells; induction of suppressive cytokines, such as IL-10 and transforming growth factor β; suppression of allergen-specific IgE and induction of IgG4 and IgA; and suppression of mast cells, basophils, eosinophils, and inflammatory dendritic cells. This review summarizes the current knowledge on the mechanisms of allergen-SIT with emphasis on the roles of Treg cells in allergen-SIT.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Administration, Cutaneous
  • Allergens / immunology
  • Antibodies / immunology
  • Antibody Specificity / immunology
  • Desensitization, Immunologic*
  • Humans
  • Hypersensitivity / immunology
  • Hypersensitivity / therapy*
  • Immune Tolerance / immunology
  • Interleukin-10 / immunology
  • Interleukin-10 / metabolism
  • T-Lymphocytes, Regulatory / immunology

Substances

  • Allergens
  • Antibodies
  • Interleukin-10