Inherited hyper-homocysteinemia as a cause of nonbacterial thrombotic endocarditis

J Heart Valve Dis. 2011 Mar;20(2):232-3.

Abstract

Background and aim of the study: Despite high rates of valve disease, non-bacterial thrombotic endocarditis remains an uncommon condition, the individual susceptibility to which might be influenced by an inherited thrombophilic state.

Methods: A comprehensive hemostasis study was conducted in a 17-year-old patient with definite nonbacterial thrombotic endocarditis, including a genetic screening for all major inherited thrombophilias.

Results: Non-bacterial thrombotic endocarditis was detected on a bicuspid aortic valve. A hemostasis work-up showed that the patient had severe hyperhomocysteinemia, and was homozygous for the C677T mutation of the methylenetetrahydrofolate reductase gene.

Conclusion: Based on the present clinical case, a novel hypothesis was proposed that the pathophysiology of non-bacterial thrombotic endocarditis might be affected by a genetic predisposition, such as an inherited thrombophilic state.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Endocarditis / blood
  • Endocarditis / etiology*
  • Hemostasis
  • Heredity
  • Homozygote
  • Humans
  • Hyperhomocysteinemia / blood
  • Hyperhomocysteinemia / complications*
  • Hyperhomocysteinemia / genetics
  • Male
  • Methylenetetrahydrofolate Reductase (NADPH2) / genetics
  • Mutation
  • Thrombosis / blood
  • Thrombosis / etiology*

Substances

  • Methylenetetrahydrofolate Reductase (NADPH2)