Exome sequencing reveals germline NPAT mutation as a candidate risk factor for Hodgkin lymphoma

Blood. 2011 Jul 21;118(3):493-8. doi: 10.1182/blood-2011-03-341560. Epub 2011 May 11.

Abstract

A strong clustering of Hodgkin lymphoma in certain families has been long acknowledged. However, the genetic factors in the background of familial Hodgkin lymphoma are largely unknown. We have studied a family of 4 cousins with a rare subtype of the disease, nodular lymphocyte predominant Hodgkin lymphoma. We applied exome sequencing together with genome-wide linkage analysis to this family and identified a truncating germline mutation in nuclear protein, ataxia-telangiectasia locus (NPAT) gene, which segregated in the family. We also studied a large number of samples from other patients with Hodgkin lymphoma, and a germline variation leading to the deletion of serine 724 was found in several cases suggesting an elevated risk for the disease (odds ratio = 4.11; P = .018). NPAT is thus far the first gene implicated in nodular lymphocyte predominant Hodgkin lymphoma predisposition.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Cycle Proteins / genetics*
  • Cell Line, Tumor
  • DNA Mutational Analysis
  • Family Health
  • Female
  • Finland / epidemiology
  • Genetic Linkage
  • Genetic Predisposition to Disease / epidemiology
  • Genetic Predisposition to Disease / genetics
  • Germ-Line Mutation*
  • Hodgkin Disease / epidemiology*
  • Hodgkin Disease / genetics*
  • Humans
  • Lymphoma, Follicular / epidemiology
  • Lymphoma, Follicular / genetics
  • Male
  • Nuclear Proteins / genetics*
  • Pedigree
  • Risk Factors
  • Young Adult

Substances

  • Cell Cycle Proteins
  • NPAT protein, human
  • Nuclear Proteins