The N-terminal env-derived amino acids of v-rel are required for full transforming activity

Virology. 1990 Jul;177(1):106-15. doi: 10.1016/0042-6822(90)90464-3.

Abstract

Expression of the v-rel oncogene of the reticuloendotheliosis virus, strain T (REV-T), can mediate the transformation of chicken spleen and bone marrow cells. Although the majority of the coding sequence of the v-rel oncogene is derived from the cellular rel sequence, the N- and C-terminal amino acids are coded for by remnants of the REV env gene. The resulting v-rel protein can be described as an env-rel-(out of frame env) fusion protein. Terminal deletion mutants were constructed to determine the role that env sequences play in the transforming activity of v-rel. Deletions were designed to remove only sequences of v-rel derived from former env sequence. Additional deletions removed more substantial amounts of coding sequence. Introduction of deleted genes into an REV-T based retroviral vector permitted the transforming activities to be determined. Deletion analysis indicated that the N-terminal region of pp59v-rel is required for the transforming activity, whereas as many as 100 C-terminal amino acids could be deleted without complete loss of the activity.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Base Sequence
  • Blotting, Western
  • Cell Line
  • Cell Transformation, Neoplastic*
  • Chromosome Deletion
  • Cloning, Molecular
  • Crossing Over, Genetic
  • DNA, Viral / genetics
  • Escherichia coli / genetics
  • Molecular Sequence Data
  • Mutation
  • Oligonucleotide Probes
  • Oncogene Proteins v-rel
  • Oncogenes*
  • Plasmids
  • Protein-Tyrosine Kinases / genetics
  • Proviruses / genetics
  • Reticuloendotheliosis virus / genetics*
  • Retroviridae / genetics*
  • Retroviridae Proteins, Oncogenic / genetics*
  • Sequence Homology, Nucleic Acid
  • Transfection
  • Viral Envelope Proteins / genetics*

Substances

  • DNA, Viral
  • Oligonucleotide Probes
  • Oncogene Proteins v-rel
  • Retroviridae Proteins, Oncogenic
  • Viral Envelope Proteins
  • Protein-Tyrosine Kinases