Granulomatous inflammation in cartilage-hair hypoplasia: risks and benefits of anti-TNF-α mAbs

J Allergy Clin Immunol. 2011 Oct;128(4):847-53. doi: 10.1016/j.jaci.2011.05.024. Epub 2011 Jun 28.

Abstract

Background: Cartilage-hair hypoplasia (CHH) is a rare autosomal recessive disorder characterized by short-limbed skeletal dysplasia. Some patients also have defects in cell-mediated immunity and antibody production. Granulomatous inflammation has been described in patients with various forms of primary immunodeficiencies but has not been reported in patients with CHH.

Objective: We sought to describe granulomatous inflammation as a novel feature in patients with CHH, assess associated immunodeficiency, and evaluate treatment options.

Methods: In a retrospective observational study we collected clinical data on 21 patients with CHH to identify and further characterize patients with granulomatous inflammation.

Results: Four unrelated patients with CHH (with variable degrees of combined immunodeficiency) had epithelioid cell granulomatous inflammation in the skin and visceral organs. Anti-TNF-α mAb therapy in 3 of these patients led to significant regression of granulomas. However, 1 treated patient had fatal progressive multifocal leukoencephalopathy caused by the JC polyomavirus. In 2 patients immune reconstitution after allogeneic hematopoietic stem cell transplantation led to the complete disappearance of granulomas.

Conclusion: To the best of our knowledge, this is the first report of granulomatous inflammation in patients with CHH. Although TNF-α antagonists can effectively suppress granulomas, the risk of severe infectious complications limits their use in immunodeficient patients.

Publication types

  • Multicenter Study

MeSH terms

  • Antibodies, Monoclonal / administration & dosage*
  • Antibodies, Monoclonal / adverse effects
  • Antibodies, Monoclonal / immunology
  • Child
  • Child, Preschool
  • Dermatitis* / immunology
  • Dermatitis* / pathology
  • Dermatitis* / therapy
  • Female
  • Granuloma* / immunology
  • Granuloma* / pathology
  • Granuloma* / therapy
  • Hair / abnormalities
  • Hair / immunology
  • Hair / pathology
  • Hematopoietic Stem Cell Transplantation
  • Hirschsprung Disease* / immunology
  • Hirschsprung Disease* / pathology
  • Hirschsprung Disease* / therapy
  • Humans
  • Immunologic Deficiency Syndromes* / immunology
  • Immunologic Deficiency Syndromes* / pathology
  • Immunologic Deficiency Syndromes* / therapy
  • Inflammation / immunology
  • Inflammation / pathology
  • Inflammation / therapy
  • Leukoencephalopathy, Progressive Multifocal / immunology
  • Leukoencephalopathy, Progressive Multifocal / pathology
  • Leukoencephalopathy, Progressive Multifocal / therapy
  • Male
  • Osteochondrodysplasias / congenital*
  • Osteochondrodysplasias / immunology
  • Osteochondrodysplasias / pathology
  • Osteochondrodysplasias / therapy
  • Primary Immunodeficiency Diseases
  • Retrospective Studies
  • Risk Factors
  • Transplantation, Homologous
  • Tumor Necrosis Factor-alpha / antagonists & inhibitors*
  • Tumor Necrosis Factor-alpha / immunology

Substances

  • Antibodies, Monoclonal
  • Tumor Necrosis Factor-alpha

Supplementary concepts

  • Cartilage-hair hypoplasia