Rab27a in pancreatic beta-cells, a busy protein in membrane trafficking

Prog Biophys Mol Biol. 2011 Nov;107(2):219-23. doi: 10.1016/j.pbiomolbio.2011.06.016. Epub 2011 Jul 7.

Abstract

The small GTPases have the 'active' GTP-bound and 'inactive' GDP-bound states, and thereby act as a molecular switch in cells. Rab27a is a member of this family and exists in T-lymphocytes, melanocytes and pancreatic beta-cells. Rab27a regulates secretion of cytolytic granules from cytotoxic T-lymphocytes and intracellular transport of melanosomes in melanocytes. In pancreatic beta-cells, Rab27a controls pre-exocytotic stages of insulin secretion. A few GTP-dependent Rab27a effectors are known to mediate these cellular functions. We recently found that Rab27a also possesses the GDP-dependent effector coronin 3. Coronin 3 regulates endocytosis in pancreatic beta-cells through its interaction with GDP-Rab27a. These results imply that GTP- and GDP-Rab27a actively regulate distinct stages in the insulin secretory pathway. In this review, we provide an overview of the roles of both GTP- and GDP-Rab27a in pancreatic beta-cells.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Animals
  • Biological Transport
  • Cell Membrane / metabolism*
  • Guanosine Diphosphate / metabolism
  • Humans
  • Insulin-Secreting Cells / cytology*
  • Insulin-Secreting Cells / metabolism
  • Microfilament Proteins / metabolism
  • rab GTP-Binding Proteins / metabolism*

Substances

  • Microfilament Proteins
  • Guanosine Diphosphate
  • rab GTP-Binding Proteins