Abstract
A 3-year-old boy receiving valproate for 1.5 months presented with sudden-onset unprovoked seizures and unconsciousness. Hypoketotic hypoglycemia, hyperammonemia, and deranged liver function were detected. Elevated medium-chain urinary acylglycines and plasma acylcarnitine were detected. His serum valproate level was elevated. Valproate toxicity had been precipitated in presence of medium-chain acyl-CoA dehydrogenase deficiency. Cranial magnetic resonance imaging brain indicated unilateral basal ganglia ischemia instead of the bilateral changes expected in metabolic disease.
Copyright © 2011 Elsevier Inc. All rights reserved.
MeSH terms
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Acyl-CoA Dehydrogenase / deficiency
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Anticonvulsants / adverse effects
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Anticonvulsants / pharmacokinetics
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Anticonvulsants / therapeutic use
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Basal Ganglia / pathology
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Brain / pathology
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Brain Ischemia / pathology
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Carnitine / analogs & derivatives
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Carnitine / blood
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Cerebral Infarction / pathology
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Child, Preschool
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Glasgow Coma Scale
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Humans
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Lipid Metabolism, Inborn Errors / pathology*
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Liver Function Tests
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Magnetic Resonance Imaging
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Male
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Respiration, Artificial
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Seizures / drug therapy
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Seizures / etiology
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Valproic Acid / adverse effects
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Valproic Acid / pharmacokinetics
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Valproic Acid / therapeutic use
Substances
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Anticonvulsants
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acylcarnitine
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Valproic Acid
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Acyl-CoA Dehydrogenase
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Carnitine
Supplementary concepts
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Medium chain acyl CoA dehydrogenase deficiency