Transposon mutagenesis with coat color genotyping identifies an essential role for Skor2 in sonic hedgehog signaling and cerebellum development

Development. 2011 Oct;138(20):4487-97. doi: 10.1242/dev.067264.

Abstract

Correct development of the cerebellum requires coordinated sonic hedgehog (Shh) signaling from Purkinje to granule cells. How Shh expression is regulated in Purkinje cells is poorly understood. Using a novel tyrosinase minigene-tagged Sleeping Beauty transposon-mediated mutagenesis, which allows for coat color-based genotyping, we created mice in which the Ski/Sno family transcriptional co-repressor 2 (Skor2) gene is deleted. Loss of Skor2 leads to defective Purkinje cell development, a severe reduction of granule cell proliferation and a malformed cerebellum. Skor2 is specifically expressed in Purkinje cells in the brain, where it is required for proper expression of Shh. Skor2 overexpression suppresses BMP signaling in an HDAC-dependent manner and stimulates Shh promoter activity, suggesting that Skor2 represses BMP signaling to activate Shh expression. Our study identifies an essential function for Skor2 as a novel transcriptional regulator in Purkinje cells that acts upstream of Shh during cerebellum development.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • Bone Morphogenetic Proteins / metabolism
  • Cell Differentiation
  • Cerebellum / abnormalities
  • Cerebellum / growth & development*
  • Cerebellum / metabolism*
  • Gene Expression Regulation, Developmental
  • Genotype
  • Hair Color / genetics
  • Hedgehog Proteins / genetics*
  • Hedgehog Proteins / metabolism*
  • Histone Deacetylases / metabolism
  • Mice
  • Mice, Knockout
  • Mice, Transgenic
  • Monophenol Monooxygenase / genetics
  • Mutagenesis, Insertional
  • Proto-Oncogene Proteins / deficiency
  • Proto-Oncogene Proteins / genetics*
  • Proto-Oncogene Proteins / metabolism*
  • Purkinje Cells / cytology
  • Purkinje Cells / metabolism
  • Repressor Proteins / deficiency
  • Repressor Proteins / genetics*
  • Repressor Proteins / metabolism*
  • Signal Transduction
  • Transforming Growth Factor beta / metabolism
  • Transposases / genetics

Substances

  • Bone Morphogenetic Proteins
  • Corl2 protein, mouse
  • Hedgehog Proteins
  • Proto-Oncogene Proteins
  • Repressor Proteins
  • Shh protein, mouse
  • Skil protein, mouse
  • Transforming Growth Factor beta
  • Monophenol Monooxygenase
  • Transposases
  • sleeping beauty transposase, human
  • Histone Deacetylases