Early use of immunosuppressives or TNF antagonists for the treatment of Crohn's disease: time for a change

Gut. 2011 Dec;60(12):1754-63. doi: 10.1136/gutjnl-2011-300934. Epub 2011 Oct 13.

Abstract

Crohn's disease is a chronic inflammatory disorder that follows a progressive and destructive course. Ultimately, uncontrolled inflammation leads to bowel damage from disease-related complications such as strictures, fistulas and abscesses and surgical resection. Conventional 'step-care', whereby corticosteroids and immunosuppressives are prescribed sequentially, is an incremental approach to treatment that does not prevent disease progression and conveys an important risk of adverse events from repeated courses of corticosteroids. Although the immunosuppressives azathioprine, 6-mercaptopurine and methotrexate are corticosteroid-sparing, they are not highly effective for inducing mucosal healing or preventing disease progression. Tumour necrosis factor antagonists induce and maintain mucosal healing and reduce surgery and hospitalisation rates. This holds out the possibility that long-term use of these agents may prevent bowel damage. Combination therapy with immunosuppressives and tumour necrosis factor antagonists is likely the best strategy for achieving optimal outcomes in patients at high risk of disease progression. However, accurate prognostic markers must be identified to guide patient selection. Long-term prospective studies with robust outcomes are still needed to establish definitively the efficacy and safety of early combination therapy to prevent bowel damage, loss of gastrointestinal tract function and permanent disability.

Publication types

  • Review

MeSH terms

  • Clinical Protocols
  • Cost-Benefit Analysis
  • Crohn Disease / diagnosis
  • Crohn Disease / drug therapy*
  • Crohn Disease / economics
  • Disease Progression
  • Drug Therapy, Combination
  • Humans
  • Immunosuppressive Agents / administration & dosage
  • Immunosuppressive Agents / adverse effects
  • Immunosuppressive Agents / economics
  • Immunosuppressive Agents / therapeutic use*
  • Infections / chemically induced
  • Lymphoma / chemically induced
  • Risk Factors
  • Tumor Necrosis Factor-alpha / antagonists & inhibitors*

Substances

  • Immunosuppressive Agents
  • Tumor Necrosis Factor-alpha