Over-expression of Sox2 in C3H10T1/2 cells inhibits osteoblast differentiation through Wnt and MAPK signalling pathways

Int Orthop. 2012 May;36(5):1087-94. doi: 10.1007/s00264-011-1368-6. Epub 2011 Oct 20.

Abstract

Purpose: Many Sox proteins play important roles both in mesoderm and ectoderm development. It is reported that Sox2, a member of this family, is essential for the maintenance of the self-renewal of embryonic stem cells (ES) and neural stem cells (NSCs). To investigate whether Sox2 participates in mesoderm development besides ectoderm, Sox2 was introduced into C3H10T1/2 cells.

Methods: We produced recombinant retrovirus expressing Sox2 in GP2-293t cells and infected the virus into C3H10T1/2 cells. Growth property, alkaline phosphatase (ALP) staining, mineralized nodules, osteogenic gene expression and related signal pathways were analysed and compared between Sox2-expressing cells and control cells.

Results: Sox2 over-expression led to increased proliferation of C3H10T1/2 cells, activation of Wnt/β-catenin and p38MAPK pathways. When cultured in osteogenic differentiation medium, ALP and mineralized nodules formation were inhibited in Sox2 over-expressing cells with down-regulation of osteogenic gene expression as well as inhibition of Wnt/β-catenin and p38MAPK pathways.

Conclusions: All these data suggested that over-expression of Sox2 promoted proliferation and inhibited osteoblast differentiation of C3H10T1/2 cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alkaline Phosphatase / metabolism
  • Animals
  • Cell Differentiation
  • Cell Line
  • Gene Expression
  • MAP Kinase Signaling System / physiology
  • Mice
  • Mitogen-Activated Protein Kinase Kinases / metabolism
  • Osteoblasts / cytology*
  • Osteoblasts / metabolism
  • Osteogenesis / genetics
  • Osteogenesis / physiology*
  • SOXB1 Transcription Factors / genetics
  • SOXB1 Transcription Factors / metabolism*
  • Signal Transduction
  • Wnt Proteins / metabolism*
  • beta Catenin / metabolism*

Substances

  • SOXB1 Transcription Factors
  • Sox2 protein, mouse
  • Wnt Proteins
  • beta Catenin
  • Mitogen-Activated Protein Kinase Kinases
  • Alkaline Phosphatase