Abstract
10-Hydroxy-trans-2-decenoic acid (10H2DA) is a major lipid component of royal jelly, a honey bee secretion used to nourish the queen bee and young larvae. In this study, we examined the effect of 10H2DA on interferon (IFN)-γ-induced nitric oxide (NO) production. IFN-γ-induced NO production and activation of the inducible NO synthase promoter were significantly inhibited by 10H2DA. IFN-γ-induced phosphorylation of signal transducer and activator of transcription-1 was not affected by 10H2DA. In contrast, IFN-γ-induced tumor necrosis factor (TNF)-α production and nuclear factor (NF)-κB activation were inhibited by 10H2DA. IFN-γ-mediated induction of interferon regulatory factor (IRF)-8, but not IRF-1, was also inhibited by 10H2DA. IFN-γ-induced TNF-α production followed by activation of NF-κB is known to be essential for NO production. Together, 10H2DA inhibited IFN-γ-induced NO production by inhibiting IRF-8 induction and TNF-α production. 10H2DA might modulate IFN-γ-mediated cellular responses by inhibiting the induction of IRF-8 and IRF-8-dependent genes.
Copyright © 2011 Elsevier Inc. All rights reserved.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Animals
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Cell Line
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Enzyme Activation / immunology
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Fatty Acids / pharmacology
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Fatty Acids, Monounsaturated / pharmacology*
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Interferon Regulatory Factors / antagonists & inhibitors*
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Interferon Regulatory Factors / genetics
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Interferon Regulatory Factors / immunology
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Interferon-gamma / antagonists & inhibitors*
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Interferon-gamma / immunology
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Macrophages / drug effects*
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Macrophages / enzymology
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Macrophages / immunology
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Mice
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NF-kappa B / immunology
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Nitric Oxide / antagonists & inhibitors
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Nitric Oxide / biosynthesis*
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Nitric Oxide Synthase Type II / antagonists & inhibitors
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Nitric Oxide Synthase Type II / genetics
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Nitric Oxide Synthase Type II / immunology
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RNA, Messenger / chemistry
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RNA, Messenger / genetics
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Reverse Transcriptase Polymerase Chain Reaction
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STAT1 Transcription Factor / immunology
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Transcription, Genetic / immunology
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Tumor Necrosis Factor-alpha / genetics
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Tumor Necrosis Factor-alpha / immunology
Substances
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Fatty Acids
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Fatty Acids, Monounsaturated
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Interferon Regulatory Factors
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NF-kappa B
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RNA, Messenger
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STAT1 Transcription Factor
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Stat1 protein, mouse
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Tumor Necrosis Factor-alpha
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interferon regulatory factor-8
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Nitric Oxide
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2-decenoic acid
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Interferon-gamma
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Nitric Oxide Synthase Type II
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royal jelly