The gut is not only the target but a source of inflammatory mediators inhibiting gastrointestinal motility during sepsis

Cell Physiol Biochem. 2011;28(4):753-60. doi: 10.1159/000335769. Epub 2011 Dec 14.

Abstract

Background: Sepsis is a common problem in intensive care patients leading to multi-organ failure and gastrointestinal paralysis.

Aim: The aim of the study was to investigate whether the gastrointestinal tract is not only the target but also the source of inflammatory mediators inhibiting gastrointestinal motility.

Methods: Mesenteric lymph was obtained from rats in which a sepsis was induced by lipopolysaccharide (LPS) intraperitoneally. Gastrointestinal motility was recorded following mesenteric lymph- or TNFα infusion into the jugular vein of separate healthy recipient rats using the strain gauge transducer technique.

Results: Infusion of sepsis lymph significantly impairs gastric and colonic motility, decreasing the motility-index in the stomach and colon by about 57% and 21% respectively in comparison to baseline motility. Among other inflammatory mediators, TNFα plays an important role in mediating the inhibitory effect of mesenteric lymph on gastrointestinal motility during sepsis.

Conclusions: The gastrointestinal tract is the source and the target of inflammatory mediators released during sepsis causing paralytic ileus.

MeSH terms

  • Animals
  • Gastrointestinal Motility / drug effects
  • Gastrointestinal Motility / physiology*
  • Inflammation Mediators / metabolism*
  • Intestinal Mucosa / metabolism
  • Intestinal Mucosa / pathology*
  • Lipopolysaccharides / toxicity
  • Lymph / metabolism
  • Male
  • Mesentery / metabolism
  • Rats
  • Rats, Sprague-Dawley
  • Sepsis / metabolism
  • Sepsis / physiopathology*
  • Tumor Necrosis Factor-alpha / metabolism

Substances

  • Inflammation Mediators
  • Lipopolysaccharides
  • Tumor Necrosis Factor-alpha