Transgenic expression of human P-selectin glycoprotein ligand-1 is not sufficient for enterovirus 71 infection in mice

Arch Virol. 2012 Mar;157(3):539-43. doi: 10.1007/s00705-011-1198-2. Epub 2011 Dec 21.

Abstract

Human PSGL-1 is a receptor for EV71 that facilitates EV71 entry and replication in mouse cells. We have evaluated the role of human PSGL-1 in EV71 infection in vivo using a transgenic mouse line. Expression of human PSGL-1 failed to enhance infectivity of clinical EV71 strains in mice; however, it promoted replication and symptom severity at an earlier stage in mice upon infection with a mouse-adapted EV71 strain. We therefore conclude that human PSGL-1 alone is not sufficient to modulate infection with the clinical EV71 strains of genotype C4 in mice.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Animals, Genetically Modified
  • Enterovirus A, Human / pathogenicity*
  • Enterovirus Infections / genetics*
  • Gene Expression*
  • Humans
  • Membrane Glycoproteins / genetics
  • Membrane Glycoproteins / metabolism*
  • Mice
  • Receptors, Virus / genetics
  • Receptors, Virus / metabolism*
  • Virus Internalization
  • Virus Replication

Substances

  • Membrane Glycoproteins
  • P-selectin ligand protein
  • Receptors, Virus