Abstract
The identification of a major class of innate immune receptors, termed pattern recognition receptors (PRRs), has boosted research on innate pathogen recognition. The immune response to a specific pathogen is not restricted to the recognition by one type of PRR or activation of a single cell type, but instead comprises complex collaborations between different receptors, cells and signal mediators. Here we will discuss the cross-talk between PRRs involved in fungal recognition, focusing on the molecular interactions occurring at the plasma membrane.
© 2012 Blackwell Publishing Ltd.
Publication types
-
Research Support, Non-U.S. Gov't
-
Review
MeSH terms
-
Animals
-
Candida albicans / immunology*
-
Candida albicans / pathogenicity
-
Candidiasis / immunology
-
Candidiasis / microbiology
-
DEAD Box Protein 58
-
DEAD-box RNA Helicases / immunology
-
Host-Pathogen Interactions*
-
Humans
-
Immunity, Innate*
-
Lectins, C-Type / immunology
-
Nod2 Signaling Adaptor Protein / immunology
-
Receptor Cross-Talk*
-
Receptors, Immunologic
-
Signal Transduction
-
Toll-Like Receptors / immunology
Substances
-
Lectins, C-Type
-
NOD2 protein, human
-
Nod2 Signaling Adaptor Protein
-
Receptors, Immunologic
-
Toll-Like Receptors
-
RIGI protein, human
-
DEAD Box Protein 58
-
DEAD-box RNA Helicases