Evidence of gene-environment interaction for the RUNX2 gene and environmental tobacco smoke in controlling the risk of cleft lip with/without cleft palate

Birth Defects Res A Clin Mol Teratol. 2012 Feb;94(2):76-83. doi: 10.1002/bdra.22885. Epub 2012 Jan 12.

Abstract

This study examined the association between 49 markers in the Runt-related transcription factor 2 (RUNX2) gene and nonsyndromic cleft lip with/without cleft palate (CL/P) among 326 Chinese case-parent trios, while considering gene-environment (GxE) interaction and parent-of-origin effects. Five single-nucleotide polymorphisms (SNPs) showed significant evidence of linkage and association with CL/P and these results were replicated in an independent European sample of 825 case-parent trios. We also report compelling evidence for interaction between markers in RUNX2 and environmental tobacco smoke (ETS). Although most marginal SNP effects (i.e., ignoring maternal exposures) were not statistically significant, eight SNPs were significant when considering possible interaction with ETS when testing for gene (G) and GxE interaction simultaneously or when considering GxE alone. Independent samples from European populations showed consistent evidence of significant GxETS interaction at two SNPs (rs6904353 and rs7748231). Our results suggest genetic variation in RUNX2 may influence susceptibility to CL/P through interacting with ETS.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, N.I.H., Intramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Asian People / genetics
  • China
  • Cleft Lip / ethnology
  • Cleft Lip / genetics*
  • Cleft Palate / ethnology
  • Cleft Palate / genetics*
  • Core Binding Factor Alpha 1 Subunit / genetics*
  • Europe
  • Female
  • Gene-Environment Interaction*
  • Genetic Linkage
  • Genetic Predisposition to Disease
  • Genotype
  • Humans
  • Male
  • Maternal Exposure
  • Polymorphism, Single Nucleotide*
  • Pregnancy
  • Prenatal Exposure Delayed Effects
  • Tobacco Smoke Pollution / adverse effects*
  • White People / genetics

Substances

  • Core Binding Factor Alpha 1 Subunit
  • RUNX2 protein, human
  • Tobacco Smoke Pollution