High expression of microRNA-210 is an independent factor indicating a poor prognosis in Japanese triple-negative breast cancer patients

Jpn J Clin Oncol. 2012 Apr;42(4):256-63. doi: 10.1093/jjco/hys001. Epub 2012 Feb 9.

Abstract

Objective: MicroRNAs have emerged as a new class of non-coding genes involved in regulating cell proliferation, differentiation and viability. Recent studies have identified miR-210 as one of a set of hypoxia-regulated microRNAs and demonstrated a direct regulatory role of HIF-1 alpha for its transcription. Here, we assessed miR-210 expression in Japanese triple-negative breast cancers and determined its clinical significance.

Methods: TaqMan MicroRNA assays for miR-210 expression were performed on 161 samples of Japanese breast cancer tissue (58 triple-negative breast cancer and 103 estrogen receptor positive/HER2 negative). Correlations between miR-210 expression and clinicopathological factors were analyzed. The effects of several variables on survival were tested by a Cox proportional hazards regression analysis.

Results: miR-210 expression in triple-negative breast cancers was significantly higher than in estrogen receptor-positive/HER2-negative breast cancers (P < 0.001). Patients whose triple-negative breast cancers showed low miR-210 expression experienced significantly better disease-free and overall survival than those with high miR-210 expression (P = 0.02 and P = 0.05, respectively). Although the prognosis of patients with triple-negative breast cancers is poor, Cox univariate and multivariate analyses demonstrated that a higher expression of miR-210 was an independent factor indicating a worse prognosis than for patients with a low level of miR-210.

Conclusions: The degree of miR-210 expression might be a clinically useful prognostic factor for decision-making regarding treatment in the adjuvant setting, especially in node-negative triple-negative breast cancer patients.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Asian People
  • Breast Neoplasms / diagnosis*
  • Breast Neoplasms / genetics*
  • Breast Neoplasms / metabolism
  • Breast Neoplasms / mortality
  • Disease-Free Survival
  • Eukaryotic Initiation Factor-3 / analysis
  • Female
  • Humans
  • MicroRNAs / metabolism*
  • Middle Aged
  • Prognosis
  • Receptor, ErbB-2 / metabolism
  • Receptors, Estrogen / metabolism
  • Receptors, Progesterone / metabolism

Substances

  • EIF3A protein, human
  • Eukaryotic Initiation Factor-3
  • MIRN210 microRNA, human
  • MicroRNAs
  • Receptors, Estrogen
  • Receptors, Progesterone
  • Receptor, ErbB-2