B8.7 antigen expression on B-CLL cells and its relationship to the LMW-BCGF responsiveness

Leuk Res. 1990;14(9):809-14. doi: 10.1016/0145-2126(90)90075-k.

Abstract

In this work, we studied the expression of B8.7 antigen on B lymphocytes from patients suffering from B type chronic lymphocytic leukemia (B-CLL) as well as on non Hodgkin lymphoma cells (NHL). B8.7 is an activation marker, which has been reported to be associated with the capacity of activated B cells to respond to LMW-BCGF. B lymphocytes of 11 out of 22 patients tested were B8.7 positive. With the exception of one case, LMW-BCGF is able to induce DNA synthesis by these cells in the absence of costimulation by anti-mu antibodies (anti-mu Ab). The LMW-BCGF dependent proliferation of these malignant cells is inhibited by the anti-B8.7 monoclonal antibody (anti-B8.7 MoAb), in the same line as that of normal B cells. These results obtained with monoclonal B cells confirm that the B8.7 molecule is involved in the signalling pathway of the LMW-BCGF.

MeSH terms

  • Aged
  • Antibodies, Monoclonal / immunology
  • Antigens, Differentiation, B-Lymphocyte / analysis
  • B-Lymphocytes / drug effects
  • B-Lymphocytes / immunology*
  • Biomarkers, Tumor / analysis*
  • Humans
  • Interleukin-2 / pharmacology
  • Interleukin-4 / pharmacology*
  • Leukemia, Lymphocytic, Chronic, B-Cell / immunology*
  • Lymphoma, Non-Hodgkin / immunology
  • Middle Aged
  • Molecular Weight

Substances

  • Antibodies, Monoclonal
  • Antigens, Differentiation, B-Lymphocyte
  • B8.7 antigen, human
  • Biomarkers, Tumor
  • Interleukin-2
  • Interleukin-4