Low-replicating viruses and strong anti-viral immune response associated with prolonged disease control in a superinfected HIV-1 LTNP elite controller

PLoS One. 2012;7(2):e31928. doi: 10.1371/journal.pone.0031928. Epub 2012 Feb 24.

Abstract

Objective: To study the causes for the lack of clinical progression in a superinfected HIV-1 LTNP elite controller patient.

Methodology and principal findings: We studied host genetic, virological and immunological factors associated with viral control in a SI long term non progressor elite controller (LTNP-EC). The individual contained both viruses and maintained undetectable viral loads for >20 years and he did not express any of the described host genetic polymorphisms associated with viral control. None of four full-length gp160 recombinants derived from the LTNP-EC replicated in heterologous peripheral blood mononuclear cells. CTL responses after SI were maintained in two samples separated by 9 years and they were higher in breadth and magnitude than responses seen in most of 250 treatment naïve patients and also 25 controller subjects. The LTNP-EC showed a neutralization response, against 4 of the 6 viruses analyzed, superior to other ECs.

Conclusions: The study demonstrated that a strong and sustained cellular and humoral immune response and low replicating viruses are associated with viral control in the superinfected LTNP-EC.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antiviral Agents / pharmacology*
  • CD4-Positive T-Lymphocytes / virology
  • Chemokines
  • Disease Progression
  • Genotype
  • HIV-1 / genetics*
  • Haplotypes
  • Humans
  • Immune System
  • Leukocytes, Mononuclear / virology
  • Likelihood Functions
  • Male
  • Mutation
  • Polymorphism, Genetic
  • Recombinant Proteins / metabolism
  • Time Factors
  • Viral Load
  • Virus Replication*

Substances

  • Antiviral Agents
  • Chemokines
  • Recombinant Proteins