Fluorescent in vivo detection reveals that IgE(+) B cells are restrained by an intrinsic cell fate predisposition

Immunity. 2012 May 25;36(5):857-72. doi: 10.1016/j.immuni.2012.02.009. Epub 2012 Mar 8.

Abstract

IgE antibodies may be protective in parasite immunity, but their aberrant production can lead to allergic disease and life-threatening anaphylaxis. Despite the importance of IgE regulation, few studies have directly examined the B cells that express IgE, because these cells are rare and difficult to detect. Here, we describe fluorescent IgE reporter mice and validate a flow cytometry procedure to allow sensitive and specific identification of IgE-expressing B cells in vivo. Similar to IgG1(+) cells, IgE(+) B cells differentiated into germinal center (GC) B cells and plasma cells (PCs) during primary immune responses to a T cell-dependent hapten-protein conjugate and the helminth Nippostrongylus brasiliensis. However, the participation of IgE(+) B cells in GCs was transient. IgE(+) B cells had an atypical propensity to upregulate the transcription factor Blimp-1 and undergo PC differentiation. Most IgE(+) PCs were short lived and showed reduced affinity maturation, revealing intrinsic mechanisms that restrict the IgE antibody response.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Apoptosis / immunology
  • B-Lymphocytes / cytology
  • B-Lymphocytes / immunology*
  • B-Lymphocytes / metabolism
  • Base Sequence
  • Cell Differentiation / immunology
  • Chimera / immunology
  • Chimera / metabolism
  • Female
  • Fluorescent Antibody Technique / methods*
  • Germinal Center / cytology
  • Germinal Center / immunology
  • Germinal Center / metabolism
  • Haptens / immunology
  • Haptens / metabolism
  • Immunoglobulin E / immunology*
  • Immunoglobulin E / metabolism*
  • Kinetics
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Molecular Sequence Data
  • Nippostrongylus / immunology
  • Nippostrongylus / metabolism
  • Plasma Cells / cytology
  • Plasma Cells / immunology
  • Plasma Cells / metabolism
  • Positive Regulatory Domain I-Binding Factor 1
  • T-Lymphocytes / cytology
  • T-Lymphocytes / immunology
  • T-Lymphocytes / metabolism
  • Transcription Factors / immunology
  • Transcription Factors / metabolism

Substances

  • Haptens
  • Prdm1 protein, mouse
  • Transcription Factors
  • Immunoglobulin E
  • Positive Regulatory Domain I-Binding Factor 1