Myo/Nog cells in normal, wounded and tumor-bearing skin

Exp Dermatol. 2012 Jun;21(6):466-8. doi: 10.1111/j.1600-0625.2012.01503.x.

Abstract

Murine and human skin were examined for the presence of Myo/Nog cells that were originally discovered in the chick embryo by their expression of MyoD mRNA, noggin and the G8 antigen. Myo/Nog cells are the primary source of noggin in telogen hair follicles. They are scarce within the interfollicular dermis and absent in the epidermis. Within 24 h following epidermal abrasion, Myo/Nog cells increase in number in the follicles and appear in the wound. Myo/Nog cells are also recruited to the stroma of tumors formed from v-Ras-transformed keratinocytes (Ker/Ras). Human squamous cell carcinomas and malignant melanomas contain significantly more Myo/Nog cells than basal cell carcinomas. Myo/Nog cells are distinct from macrophages, granulocytes and cells expressing alpha smooth muscle actin in the tumor stroma. Myo/Nog cells may be modulators of skin homoeostasis and wound healing, and potential diagnostic and therapeutic targets in skin cancer.

Publication types

  • Letter
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Carcinoma, Basal Cell / metabolism
  • Carcinoma, Squamous Cell / metabolism
  • Carrier Proteins / metabolism*
  • Humans
  • Melanoma / metabolism
  • Mice
  • MyoD Protein / metabolism*
  • Skin / metabolism*
  • Skin Neoplasms / metabolism*
  • Wounds and Injuries / metabolism*

Substances

  • Carrier Proteins
  • MyoD Protein
  • noggin protein