Oncogenes in multiple myeloma: point mutation of N-ras

Oncogene. 1990 Nov;5(11):1659-63.

Abstract

Alterations of ras, c-myc and bcl-1 have been described in hematologic malignancies of lymphoid origin. We investigated the structure of these genes and evaluated the frequency of point mutations involving H-, K- or N-ras in bone marrow samples from patients with multiple myeloma. No abnormalities were detected in the c-myc and bcl-1 genes, but two of 17 patients were found to have N-ras mutations by differential oligonucleotide hybridization and dideoxynucleotide sequencing following amplification by polymerase chain reaction. Bone marrow DNA from both patients had identical missense mutations of N-ras codon 61 changing CAA to AAA, resulting in a substitution of lysine for glutamine in the encoded protein. Multiple myeloma is the first mature B cell neoplasm found to harbor ras mutations.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Base Sequence
  • Bone Marrow / chemistry
  • Bone Marrow / pathology
  • Genes, ras*
  • Humans
  • Male
  • Middle Aged
  • Multiple Myeloma / drug therapy
  • Multiple Myeloma / genetics*
  • Multiple Myeloma / pathology
  • Mutation*
  • Nucleic Acid Hybridization
  • Oligonucleotide Probes
  • Oncogenes*
  • Plasma Cells / pathology
  • Polymerase Chain Reaction

Substances

  • Oligonucleotide Probes