ALSoD: A user-friendly online bioinformatics tool for amyotrophic lateral sclerosis genetics

Hum Mutat. 2012 Sep;33(9):1345-51. doi: 10.1002/humu.22157. Epub 2012 Jul 16.

Abstract

Amyotrophic lateral sclerosis (ALS) is the commonest adult onset motor neuron disease, with a peak age of onset in the seventh decade. With advances in genetic technology, there is an enormous increase in the volume of genetic data produced, and a corresponding need for storage, analysis, and interpretation, particularly as our understanding of the relationships between genotype and phenotype mature. Here, we present a system to enable this in the form of the ALS Online Database (ALSoD at http://alsod.iop.kcl.ac.uk), a freely available database that has been transformed from a single gene storage facility recording mutations in the SOD1 gene to a multigene ALS bioinformatics repository and analytical instrument combining genotype, phenotype, and geographical information with associated analysis tools. These include a comparison tool to evaluate genes side by side or jointly with user configurable features, a pathogenicity prediction tool using a combination of computational approaches to distinguish variants with nonfunctional characteristics from disease-associated mutations with more dangerous consequences, and a credibility tool to enable ALS researchers to objectively assess the evidence for gene causation in ALS. Furthermore, integration of external tools, systems for feedback, annotation by users, and two-way links to collaborators hosting complementary databases further enhance the functionality of ALSoD.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amyotrophic Lateral Sclerosis / diagnosis
  • Amyotrophic Lateral Sclerosis / genetics*
  • Computational Biology / methods*
  • Databases, Genetic*
  • Genetic Association Studies
  • Genetic Predisposition to Disease
  • Geography
  • Humans
  • Information Storage and Retrieval
  • Internet
  • Mutation
  • Phenotype
  • Superoxide Dismutase / genetics
  • Superoxide Dismutase-1
  • User-Computer Interface*

Substances

  • SOD1 protein, human
  • Superoxide Dismutase
  • Superoxide Dismutase-1