The regulation of the SARK promoter activity by hormones and environmental signals

Plant Sci. 2012 Sep:193-194:39-47. doi: 10.1016/j.plantsci.2012.05.005. Epub 2012 May 17.

Abstract

The Senescence Associated Receptor Protein Kinase (P(SARK)) promoter, fused to isopentenyltransferase (IPT) gene has been shown to promote drought tolerance in crops. We dissected P(SARK) in order to understand the various elements associated with its activation and suppression. The activity of P(SARK) was higher in mature and early senescing leaves, and abiotic stress induced its activity in mature leaves. Bioinformatics analysis suggests the interactions of multiple cis-acting elements in the control of P(SARK) activity. In vitro gel shift assays and yeast one hybrid system revealed interactions of P(SARK) with transcription factors related to abscisic acid and cytokinin response. Deletion analysis of P(SARK), fused to GUS-reporter gene was used to identify specific regions regulating transcription under senescence or during drought stress. Effects of exogenous hormonal treatments were characterized in entire plants and in leaf disk assays, and regions responsive to various hormones were defined. Our results indicate a complex interaction of plant hormones and additional factors modulating P(SARK) activity under stress resulting in a transient induction of expression.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Adaptation, Physiological / genetics
  • Aging / genetics*
  • Base Sequence
  • Droughts
  • Gene Expression Regulation, Plant
  • Gene-Environment Interaction*
  • Genes, Reporter / physiology
  • Molecular Sequence Data
  • Nicotiana / genetics
  • Nicotiana / metabolism
  • Plant Growth Regulators / metabolism*
  • Plant Leaves / genetics*
  • Plant Leaves / metabolism*
  • Plants, Genetically Modified
  • Promoter Regions, Genetic / physiology
  • Protein Kinases / metabolism
  • Transcription Factors / metabolism

Substances

  • Plant Growth Regulators
  • Transcription Factors
  • Protein Kinases