Alexander disease with mild dorsal brainstem atrophy and infantile spasms

Brain Dev. 2013 May;35(5):441-4. doi: 10.1016/j.braindev.2012.06.009. Epub 2012 Jul 20.

Abstract

We present the case of a Japanese male infant with Alexander disease who developed infantile spasms at 8 months of age. The patient had a cluster of partial seizures at 4 months of age. He presented with mild general hypotonia and developmental delay. Macrocephaly was not observed. Brain magnetic resonance imaging (MRI) findings fulfilled all MRI-based criteria for the diagnosis of Alexander disease and revealed mild atrophy of the dorsal pons and medulla oblongata with abnormal intensities. DNA analysis disclosed a novel heterozygous missense mutation (c.1154 C>T, p.S385F) in the glial fibrillary acidic protein gene. At 8 months of age, tonic spasms occurred, and electroencephalography (EEG) revealed hypsarrhythmia. Lamotrigine effectively controlled the infantile spasms and improved the abnormal EEG findings. Although most patients with infantile Alexander disease have epilepsy, infantile spasms are rare. This comorbid condition may be associated with the distribution of the brain lesions and the age at onset of Alexander disease.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alexander Disease / complications*
  • Alexander Disease / drug therapy
  • Alexander Disease / genetics
  • Alexander Disease / pathology*
  • Anticonvulsants / therapeutic use
  • Atrophy / complications
  • Atrophy / genetics
  • Brain Stem / pathology*
  • Brain Waves / physiology
  • Corpus Callosum / pathology
  • DNA Mutational Analysis
  • Electroencephalography
  • Glial Fibrillary Acidic Protein / genetics
  • Humans
  • Infant
  • Magnetic Resonance Imaging
  • Male
  • Spasms, Infantile / complications*
  • Spasms, Infantile / drug therapy
  • Spasms, Infantile / genetics
  • Valproic Acid / therapeutic use

Substances

  • Anticonvulsants
  • Glial Fibrillary Acidic Protein
  • Valproic Acid