The Nlrp3 inflammasome promotes age-related thymic demise and immunosenescence

Cell Rep. 2012 Jan 26;1(1):56-68. doi: 10.1016/j.celrep.2011.11.005. Epub 2012 Jan 26.

Abstract

The collapse of thymic stromal cell microenvironment with age and resultant inability of the thymus to produce naive T cells contributes to lower immune-surveillance in the elderly. Here we show that age-related increase in 'lipotoxic danger signals' such as free cholesterol (FC) and ceramides, leads to thymic caspase-1 activation via the Nlrp3 inflammasome. Elimination of Nlrp3 and Asc, a critical adaptor required for inflammasome assembly, reduces age-related thymic atrophy and results in an increase in cortical thymic epithelial cells, T cell progenitors and maintenance of T cell repertoire diversity. Using a mouse model of irradiation and hematopoietic stem cell transplantation (HSCT), we show that deletion of the Nlrp3 inflammasome accelerates T cell reconstitution and immune recovery in middle-aged animals. Collectively, these data demonstrate that lowering inflammasome-dependent caspase-1 activation increases thymic lymphopoiesis and suggest that Nlrp3 inflammasome inhibitors may aid the re-establishment of a diverse T cell repertoire in middle-aged or elderly patients undergoing HSCT.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aging / drug effects
  • Aging / immunology*
  • Animals
  • Apoptosis / drug effects
  • Apoptosis / immunology
  • Carrier Proteins / metabolism*
  • Caspase 1 / metabolism
  • Cellular Microenvironment
  • Cellular Senescence / drug effects
  • Cellular Senescence / immunology
  • Ceramides / metabolism
  • Cholesterol / metabolism
  • Enzyme Activation / drug effects
  • Epithelial Cells / drug effects
  • Epithelial Cells / immunology
  • Epithelial Cells / pathology
  • Gene Deletion
  • Hematopoietic Stem Cell Transplantation
  • Inflammasomes / metabolism*
  • Interleukin-18 / metabolism
  • Interleukin-1beta / metabolism
  • Lipids / toxicity
  • Mice
  • NLR Family, Pyrin Domain-Containing 3 Protein
  • Organ Size / drug effects
  • Stem Cells / drug effects
  • Stem Cells / pathology
  • T-Lymphocytes / drug effects
  • T-Lymphocytes / pathology
  • Thymocytes / drug effects
  • Thymocytes / immunology
  • Thymocytes / pathology
  • Thymus Gland / enzymology
  • Thymus Gland / growth & development*
  • Thymus Gland / immunology*
  • Thymus Gland / pathology

Substances

  • Carrier Proteins
  • Ceramides
  • Inflammasomes
  • Interleukin-18
  • Interleukin-1beta
  • Lipids
  • NLR Family, Pyrin Domain-Containing 3 Protein
  • Nlrp3 protein, mouse
  • Cholesterol
  • Caspase 1