Myocardial Notch signaling reprograms cardiomyocytes to a conduction-like phenotype

Circulation. 2012 Aug 28;126(9):1058-66. doi: 10.1161/CIRCULATIONAHA.112.103390. Epub 2012 Jul 26.

Abstract

Background: Notch signaling has previously been shown to play an essential role in regulating cell fate decisions and differentiation during cardiogenesis in many systems including Drosophila, Xenopus, and mammals. We hypothesized that Notch may also be involved in directing the progressive lineage restriction of cardiomyocytes into specialized conduction cells.

Methods and results: In hearts where Notch signaling is activated within the myocardium from early development onward, Notch promotes a conduction-like phenotype based on ectopic expression of conduction system-specific genes and cell autonomous changes in electrophysiology. With the use of an in vitro assay to activate Notch in newborn cardiomyocytes, we observed global changes in the transcriptome, and in action potential characteristics, consistent with reprogramming to a conduction-like phenotype.

Conclusions: Notch can instruct the differentiation of chamber cardiac progenitors into specialized conduction-like cells. Plasticity remains in late-stage cardiomyocytes, which has potential implications for engineering of specialized cardiovascular tissues.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Action Potentials
  • Adenoviridae / genetics
  • Animals
  • Animals, Newborn
  • Atrioventricular Node / cytology*
  • Basic Helix-Loop-Helix Transcription Factors / biosynthesis
  • Basic Helix-Loop-Helix Transcription Factors / genetics
  • Cell Lineage
  • Contactin 2 / biosynthesis
  • Contactin 2 / genetics
  • Gene Expression Regulation, Developmental*
  • Homeobox Protein Nkx-2.5
  • Homeodomain Proteins / biosynthesis
  • Homeodomain Proteins / genetics
  • Mice
  • Myocardium / metabolism*
  • Myocytes, Cardiac / metabolism*
  • Myocytes, Cardiac / ultrastructure
  • NAV1.5 Voltage-Gated Sodium Channel
  • Neuronal Plasticity
  • Patch-Clamp Techniques
  • Phenotype
  • Purkinje Fibers / cytology
  • Receptor, Notch1 / genetics
  • Receptor, Notch1 / physiology*
  • Recombinant Fusion Proteins / physiology
  • Signal Transduction / physiology
  • Sodium Channels / biosynthesis
  • Sodium Channels / genetics
  • T-Box Domain Proteins / biosynthesis
  • T-Box Domain Proteins / genetics
  • Transcription Factor HES-1
  • Transcription Factors / biosynthesis
  • Transcription Factors / genetics

Substances

  • Basic Helix-Loop-Helix Transcription Factors
  • Cntn2 protein, mouse
  • Contactin 2
  • Hes1 protein, mouse
  • Homeobox Protein Nkx-2.5
  • Homeodomain Proteins
  • NAV1.5 Voltage-Gated Sodium Channel
  • Nkx2-5 protein, mouse
  • Notch1 protein, mouse
  • Receptor, Notch1
  • Recombinant Fusion Proteins
  • Scn5a protein, mouse
  • Sodium Channels
  • T-Box Domain Proteins
  • Tbx3 protein, mouse
  • Transcription Factor HES-1
  • Transcription Factors