Abstract
γδ T cells respond rapidly to keratinocyte damage, providing essential contributions to the skin wound healing process. The molecular interactions regulating their response are unknown. Here, we identify a role for interaction of plexin B2 with the CD100 receptor in epithelial repair. In vitro blocking of plexin B2 or CD100 inhibited γδ T cell activation. Furthermore, CD100 deficiency in vivo resulted in delayed repair of cutaneous wounds due to a disrupted γδ T cell response to keratinocyte damage. Ligation of CD100 in γδ T cells induced cellular rounding via signals through ERK kinase and cofilin. Defects in this rounding process were evident in the absence of CD100-mediated signals, thereby providing a mechanistic explanation for the defective wound healing in CD100-deficient animals. The discovery of immune functions for plexin B2 and CD100 provides insight into the complex cell-cell interactions between epithelial resident γδ T cells and the neighboring cells they support.
Copyright © 2012 Elsevier Inc. All rights reserved.
Publication types
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Research Support, N.I.H., Extramural
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Research Support, Non-U.S. Gov't
MeSH terms
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Actin Depolymerizing Factors / metabolism
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Animals
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Antigens, CD / immunology*
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Antigens, CD / metabolism
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CHO Cells
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Cell Communication / immunology
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Cell Shape
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Cricetinae
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Epidermis / immunology
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Epidermis / injuries
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Extracellular Signal-Regulated MAP Kinases / metabolism
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HEK293 Cells
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Humans
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Keratinocytes / immunology
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Keratinocytes / metabolism
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Langerhans Cells / immunology*
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Langerhans Cells / metabolism
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Lymphocyte Activation / immunology
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Mass Spectrometry
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Mice
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Mice, Inbred C57BL
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Mice, Transgenic
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Nerve Tissue Proteins / immunology*
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Nerve Tissue Proteins / metabolism
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Phosphorylation
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Protein Binding / immunology
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Receptors, Antigen, T-Cell, gamma-delta / immunology*
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Receptors, Antigen, T-Cell, gamma-delta / metabolism
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Semaphorins / immunology*
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Semaphorins / metabolism
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Sequence Analysis, Protein
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Surface Plasmon Resonance
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T-Lymphocytes / immunology*
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T-Lymphocytes / metabolism
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Wound Healing / immunology
Substances
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Actin Depolymerizing Factors
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Antigens, CD
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CD100 antigen
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Nerve Tissue Proteins
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Plxnb2 protein, mouse
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Receptors, Antigen, T-Cell, gamma-delta
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Semaphorins
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Extracellular Signal-Regulated MAP Kinases