A novel deletion in SMPX causes a rare form of X-linked progressive hearing loss in two families due to a founder effect

Hum Mutat. 2013 Jan;34(1):66-9. doi: 10.1002/humu.22205. Epub 2012 Oct 11.

Abstract

X-linked hearing loss is the rarest form of genetic hearing loss contributing to <1% of cases. We identified a multiplex family from Newfoundland (Family 2024) segregating X-linked hearing loss. Haplotyping of the X chromosome and sequencing of positional candidate genes revealed a novel point deletion (c.99delC) in SMPX which encodes a small muscle protein responsible for reducing mechanical stress during muscle contraction. This novel deletion causes a frameshift and a premature stop codon (p.Arg34GlufsX47). We successfully sequenced both SMPX wild-type and mutant alleles from cDNA of a lymphoblastoid cell line, suggesting that the mutant allele may not be degraded via nonsense-mediated mRNA decay. To investigate the role of SMPX in other subpopulations, we fully sequenced SMPX in 229 Canadian probands with hearing loss and identified a second Newfoundland Family (2196) with the same mutation, and a shared haplotype on the X chromosome, suggesting a common ancestor.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Base Sequence
  • DNA Mutational Analysis
  • Exons / genetics
  • Family Health
  • Female
  • Founder Effect*
  • Genetic Diseases, X-Linked / genetics*
  • Hearing Loss / genetics*
  • Humans
  • Male
  • Muscle Proteins / genetics*
  • Pedigree
  • Sequence Deletion*

Substances

  • Muscle Proteins
  • SMPX protein, human