Potent neutralization of botulinum neurotoxin/B by synergistic action of antibodies recognizing protein and ganglioside receptor binding domain

PLoS One. 2012;7(8):e43845. doi: 10.1371/journal.pone.0043845. Epub 2012 Aug 29.

Abstract

Background: Botulinum neurotoxins (BoNTs), the causative agents for life-threatening human disease botulism, have been recognized as biological warfare agents. Monoclonal antibody (mAb) therapeutics hold considerable promise as BoNT therapeutics, but the potencies of mAbs against BoNTs are usually less than that of polyclonal antibodies (or oligoclonal antibodies). The confirmation of key epitopes with development of effective mAb is urgently needed.

Methods and findings: We selected 3 neutralizing mAbs which recognize different non-overlapping epitopes of BoNT/B from a panel of neutralizing antibodies against BoNT/B. By comparing the neutralizing effects among different combination groups, we found that 8E10, response to ganglioside receptor binding site, could synergy with 5G10 and 2F4, recognizing non-overlapping epitopes within Syt II binding sites. However, the combination of 5G10 with 2F4 blocking protein receptor binding sites did not achieve synergistical effects. Moreover, we found that the binding epitope of 8E10 was conserved among BoNT A, B, E, and F, which might cross-protect the challenge of different serotypes of BoNTs in vivo.

Conclusions: The combination of two mAbs recognizing different receptors' binding domain in BoNTs has a synergistic effect. 8E10 is a potential universal partner for the synergistical combination with other mAb against protein receptor binding domain in BoNTs of other serotypes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Antibodies, Monoclonal / immunology*
  • Antibodies, Neutralizing / immunology*
  • Antibody Specificity
  • Binding Sites
  • Botulinum Toxins / chemistry
  • Botulinum Toxins / immunology*
  • Botulinum Toxins / metabolism
  • Botulinum Toxins / toxicity
  • Botulinum Toxins, Type A
  • Conserved Sequence
  • Epitope Mapping
  • Female
  • Mice
  • Mice, Inbred BALB C
  • Models, Molecular
  • Molecular Sequence Data
  • Protein Structure, Tertiary
  • Receptors, Cell Surface / metabolism*

Substances

  • Antibodies, Monoclonal
  • Antibodies, Neutralizing
  • Receptors, Cell Surface
  • ganglioside receptor
  • rimabotulinumtoxinB
  • Botulinum Toxins
  • Botulinum Toxins, Type A

Grants and funding

This work was supported by grants from National Nature Science Foundation for China, Ministry of Science & Technology of China (973 and 863 Program Projects), Shanghai Commission of Science & Technology (Key Laboratory and projects) as well as special program projects for infectious diseases and new drug development from Ministry of Science & Technology of China (2010ZX09401-407). Dr. Jianxin Dai is recipient of Pujiang Scholar Award from Shanghai Commission of Education. The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.