Association between the CD14 gene C-159T polymorphism and serum soluble CD14 with pulmonary tuberculosis

Int J Tuberc Lung Dis. 2012 Oct;16(10):1383-7. doi: 10.5588/ijtld.11.0827.

Abstract

Background: Functional C-159T polymorphism in the promoter region of the CD14 lipopolysaccharide receptor has been reported to be associated with the development of tuberculosis (TB).

Objective: To assess the association of CD14 C-159T polymorphism and serum soluble CD14 (sCD14) levels with pulmonary tuberculosis (TB) in an Iranian population living in a TB-endemic area.

Design: A case-control study was performed prospectively on 120 newly diagnosed pulmonary TB patients and 131 healthy subjects. C-159T polymorphism was performed using amplification refractory mutation system polymerase chain reaction (ARMS-PCR). Concentrations of sCD14 were measured in serum samples using enzyme-linked immunosorbent assay.

Results: The genotype frequencies of C-159T polymorphism differed significantly between TB patients and controls (P = 0.006). The risk of TB was 2.3-fold greater in individuals with the T-allele (CT + TT) in comparison to those without (OR 2.3, 95%CI 1.2-4.3, P = 0.006). Mean total sCD14 was significantly increased in the serum of patients with newly diagnosed pulmonary TB (mean ± SD = 3177 ± 751 ng/ml) compared to healthy controls (mean ± SD = 2955 ± 424 ng/ml, P < 0.004).

Conclusion: These data indicate that the C-159T polymorphism of the CD14 gene is associated with TB; serum sCD14 levels were higher in TB patients in a sample of the Iranian population.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Biomarkers / blood
  • DNA / genetics*
  • Female
  • Flow Cytometry
  • Gene Frequency
  • Genetic Predisposition to Disease*
  • Genotype
  • Humans
  • Iran / epidemiology
  • Lipopolysaccharide Receptors / blood
  • Lipopolysaccharide Receptors / genetics*
  • Lipopolysaccharide Receptors / immunology
  • Male
  • Middle Aged
  • Polymerase Chain Reaction
  • Polymorphism, Genetic*
  • Prevalence
  • Retrospective Studies
  • Tuberculosis, Pulmonary / epidemiology
  • Tuberculosis, Pulmonary / genetics*
  • Tuberculosis, Pulmonary / immunology

Substances

  • Biomarkers
  • Lipopolysaccharide Receptors
  • DNA