Identification of inhibitors against interaction between pro-inflammatory sPLA2-IIA protein and integrin αvβ3

Bioorg Med Chem Lett. 2013 Jan 1;23(1):340-5. doi: 10.1016/j.bmcl.2012.10.080. Epub 2012 Oct 24.

Abstract

Increased concentrations of secreted phospholipase A2 type IIA (sPLA2-IIA), have been found in the synovial fluid of patients with rheumatoid arthritis. It has been shown that sPLA2-IIA specifically binds to integrin αvβ3, and initiates a signaling pathway that leads to cell proliferation and inflammation. Therefore, the interaction between integrin and sPLA2-IIA could be a potential therapeutic target for the treatment of proliferation or inflammation-related diseases. Two one-bead-one-compound peptide libraries were constructed and screened, and seven target hits were identified. Herein we report the identification, synthesis, and biological testing of two pyrazolylthiazole-tethered peptide hits and their analogs. Biological assays showed that these compounds were able to suppress the sPLA2-IIA-integrin interaction and sPLA2-IIA-induced migration of monocytic cells and that the blockade of the sPLA2-IIA-integrin binding was specific to sPLA2-IIA and not to the integrin.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Amino Acid Sequence
  • Binding Sites
  • Group II Phospholipases A2 / antagonists & inhibitors
  • Group II Phospholipases A2 / metabolism*
  • Integrin alphaVbeta3 / antagonists & inhibitors
  • Integrin alphaVbeta3 / metabolism*
  • Molecular Docking Simulation
  • Peptide Library
  • Peptides / chemistry
  • Peptides / pharmacology
  • Protein Binding / drug effects
  • Protein Structure, Tertiary
  • Structure-Activity Relationship
  • Thiazoles / chemistry

Substances

  • Integrin alphaVbeta3
  • Peptide Library
  • Peptides
  • Thiazoles
  • Group II Phospholipases A2