Association of the CCR5Δ32 polymorphism and its ligand RANTES-403G/A polymorphism with coronary artery disease: a meta-analysis

Thromb Res. 2013 Mar;131(3):e77-84. doi: 10.1016/j.thromres.2012.07.024. Epub 2013 Jan 9.

Abstract

Introduction: To explore the relationship between polymorphisms in the RANTES and CCR5 genes and the risk of coronary artery disease (CAD).

Materials and methods: We conducted a meta-analysis on two genetic variants (RANTES-403G/A and CCR5Δ32). Publication bias was tested by the Egger's regression test and Begg's test. Sensitivity analysis and subgroup analyses were performed to explore the heterogeneity among studies.

Results: No significant association of RANTES-403G/A polymorphism and CAD risk was observed (dominant model: RR=1.02, 95%CI=0.90-1.06; recessive model: RR=1.27, 95%CI=0.90-1.80). However, after excluding the study conducted by Yangsoo et al., the pooled relative ratio (RR) in the dominant model suggested that the RANTES-403G/A polymorphism was positively associated with CAD risk. The subgroup analyses found that a positive relationship between the polymorphism and CAD risk was restricted to the Caucasian population. A meta-analysis of studies on the CCR5Δ32 polymorphism showed no significant association with CAD risk both in dominant (RR=1.05, 95%CI=0.92-1.21) and recessive (RR=1.27, 95%CI=0.90-1.80) models. Moreover, no association was identified in the subgroup analyses.

Conclusions: The RANTES-403G/A polymorphism is not associated with CAD risk, but does most likely increase CAD risk in Caucasians. Moreover, no relationship between the CCR5∆32 polymorphism and risk of CAD was found.

Publication types

  • Meta-Analysis

MeSH terms

  • Adult
  • Aged
  • Case-Control Studies
  • Chemokine CCL5 / genetics*
  • Coronary Artery Disease / genetics*
  • Female
  • Genetic Predisposition to Disease
  • Genetic Variation
  • Genotype
  • Humans
  • Male
  • Middle Aged
  • Polymorphism, Genetic*
  • Receptors, CCR5 / genetics*
  • Risk Factors
  • Sensitivity and Specificity

Substances

  • CCL5 protein, human
  • Chemokine CCL5
  • Receptors, CCR5